Literature DB >> 18071141

Multicenter assessment of CSF-phosphorylated tau for the prediction of conversion of MCI.

M Ewers1, K Buerger, S J Teipel, P Scheltens, J Schröder, R P Zinkowski, F H Bouwman, P Schönknecht, N S M Schoonenboom, N Andreasen, A Wallin, J F DeBernardis, D J Kerkman, B Heindl, K Blennow, H Hampel.   

Abstract

BACKGROUND: The measurement of hyperphosphorylated tau (p-tau) in CSF has been proposed as a biomarker candidate for the prediction of Alzheimer disease (AD) in patients with mild cognitive impairment (MCI). However, a standard quantitative criterion of p-tau has not been evaluated.
OBJECTIVE: To assess in a multicenter study the predictive accuracy of an a priori defined criterion of tau phosphorylated at threonine 231 (p-tau(231)) for the prediction of conversion from MCI to AD during a short-term observation interval.
METHODS: The study included 43 MCI converters, 45 stable MCI (average follow-up interval = 1.5 years), and 57 healthy controls (at baseline only). Subjects were recruited at four international expert sites in a retrospective study design. Cox regression models stratified according to center were used to predict conversion status. Bootstrapped 95% CIs of classification accuracy were computed.
RESULTS: Levels of p-tau(231) were a significant predictor of conversion (B = 0.026, p = 0.001), independent of age, gender, Mini-Mental State Examination, and ApoE genotype. For an a priori-defined cutoff point (27.32 pg/mL), sensitivity ranged between 66.7 and 100% and specificity between 66.7 and 77.8% among centers. The bootstrapped mean percentage of correctly classified cases was 79.95% (95% CI = 79.9 to 80.00%). Post hoc defined cutoff values yielded a mean bootstrapped classification accuracy of 80.45% (95% CI = 80.24 to 80.76%).
CONCLUSIONS: An a priori defined cutoff value of p-tau(231) yields relatively stable results across centers, suggesting a good feasibility of a standard criterion of p-tau(231) for the prediction of Alzheimer disease.

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Year:  2007        PMID: 18071141     DOI: 10.1212/01.wnl.0000286944.22262.ff

Source DB:  PubMed          Journal:  Neurology        ISSN: 0028-3878            Impact factor:   9.910


  48 in total

1.  Alzheimer disease pathology in subjects without dementia in 2 studies of aging: the Nun Study and the Adult Changes in Thought Study.

Authors:  Karen S SantaCruz; Joshua A Sonnen; Maryam Kherad Pezhouh; Mark F Desrosiers; Peter T Nelson; Suzanne L Tyas
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2.  CSF phospho-tau correlates with behavioural decline and brain insoluble phospho-tau levels in a rat model of tauopathy.

Authors:  Norbert Zilka; Miroslava Korenova; Branislav Kovacech; Khalid Iqbal; Michal Novak
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3.  Age and diagnostic performance of Alzheimer disease CSF biomarkers.

Authors:  N Mattsson; E Rosén; O Hansson; N Andreasen; L Parnetti; M Jonsson; S-K Herukka; W M van der Flier; M A Blankenstein; M Ewers; K Rich; E Kaiser; M M Verbeek; M Olde Rikkert; M Tsolaki; E Mulugeta; D Aarsland; P J Visser; J Schröder; J Marcusson; M de Leon; H Hampel; P Scheltens; A Wallin; M Eriksdotter-Jönhagen; L Minthon; B Winblad; K Blennow; H Zetterberg
Journal:  Neurology       Date:  2012-02-01       Impact factor: 9.910

4.  Tau as a biomarker of neurodegenerative diseases.

Authors:  Susanna Schraen-Maschke; Nicolas Sergeant; Claire-Marie Dhaenens; Stéphanie Bombois; Vincent Deramecourt; Marie-Laure Caillet-Boudin; Florence Pasquier; Claude-Alain Maurage; Bernard Sablonnière; Eugeen Vanmechelen; Luc Buée
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5.  Biomarkers for the identification and treatment of dementia.

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Review 6.  Biomarkers for Alzheimer disease in cerebrospinal fluid, urine, and blood.

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Journal:  Mol Diagn Ther       Date:  2008       Impact factor: 4.074

7.  Tau is reduced in AD plasma and validation of employed ELISA methods.

Authors:  D Larry Sparks; Richard J Kryscio; Marwan N Sabbagh; Chuck Ziolkowski; Yushun Lin; Lisa M Sparks; Carolyn Liebsack; Sherry Johnson-Traver
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8.  Pittsburgh compound B imaging and prediction of progression from cognitive normality to symptomatic Alzheimer disease.

Authors:  John C Morris; Catherine M Roe; Elizabeth A Grant; Denise Head; Martha Storandt; Alison M Goate; Anne M Fagan; David M Holtzman; Mark A Mintun
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9.  Hypothetical model of dynamic biomarkers of the Alzheimer's pathological cascade.

Authors:  Clifford R Jack; David S Knopman; William J Jagust; Leslie M Shaw; Paul S Aisen; Michael W Weiner; Ronald C Petersen; John Q Trojanowski
Journal:  Lancet Neurol       Date:  2010-01       Impact factor: 44.182

10.  Nicotinamide restores cognition in Alzheimer's disease transgenic mice via a mechanism involving sirtuin inhibition and selective reduction of Thr231-phosphotau.

Authors:  Kim N Green; Joan S Steffan; Hilda Martinez-Coria; Xuemin Sun; Steven S Schreiber; Leslie Michels Thompson; Frank M LaFerla
Journal:  J Neurosci       Date:  2008-11-05       Impact factor: 6.167

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