Literature DB >> 18036925

Ventricular restraint prevents infarct expansion and improves borderzone function after myocardial infarction: a study using magnetic resonance imaging, three-dimensional surface modeling, and myocardial tagging.

Aaron S Blom1, James J Pilla, Jeffrey Arkles, Larry Dougherty, Liam P Ryan, Joseph H Gorman, Michael A Acker, Robert C Gorman.   

Abstract

BACKGROUND: Infarct expansion is associated with impaired borderzone function, adverse remodeling, and poor long-term prognosis. We hypothesized that left ventricular restraint early after myocardial infarction limits infarct expansion, preserves borderzone function, and reduces remodeling.
METHODS: We used an ovine model as well as high spatial and temporal resolution cardiac magnetic resonance imaging to quantify total and infarcted left ventricular epicardial surface area at baseline and 1 week and 12 weeks after anterior wall infarction in 10 animals. Five animals were randomly assigned to treatment with left ventricular restraint (Acorn cardiac support device) 1 week after infarction. Five animals were untreated controls. Total left ventricular surface area was measured by importing the end-diastolic magnetic resonance imaging-derived epicardial contours into custom software, which creates a three-dimensional surface from the two-dimensional magnetic resonance imaging contours. Infarct area was calculated from magnetic resonance imaging-detectable titanium markers placed at the infarct border. Borderzone radial and circumferential strains during systole were also assessed using myocardial tagging techniques as a measure of contractile function.
RESULTS: The infarct area 1 week after infarction was 1,177 +/- 386 mm(2) in the control group and 1,124 +/- 427 mm(2) in the cardiac support device group. After 12 weeks, infarct area was 3,666 +/- 1,013 mm(2) in the control group and 1,227 +/- 301 mm(2) in the cardiac support device group. Borderzone systolic radial strain decreased from 12.6% +/- 0.77% to 3.6% +/- 0.3% after infarction in the control group and 13.7% +/- 0.87% to 4.7% +/- 0.3% in the cardiac support device group. At 12 weeks after infarction, radial strain was 3.4% +/- 0.5% in the control group and 6.7% +/- 0.4% in the cardiac support device group.
CONCLUSIONS: Early postinfarction left ventricular restraint limits infarct expansion and improves borderzone contractile function.

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Mesh:

Year:  2007        PMID: 18036925     DOI: 10.1016/j.athoracsur.2007.06.062

Source DB:  PubMed          Journal:  Ann Thorac Surg        ISSN: 0003-4975            Impact factor:   4.330


  22 in total

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3.  Rotating frame spin lattice relaxation in a swine model of chronic, left ventricular myocardial infarction.

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8.  Allogeneic mesenchymal precursor cell therapy to limit remodeling after myocardial infarction: the effect of cell dosage.

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Journal:  Sci Transl Med       Date:  2014-02-12       Impact factor: 17.956

10.  Preclinical evaluation of the engineered stem cell chemokine stromal cell-derived factor 1α analog in a translational ovine myocardial infarction model.

Authors:  John W Macarthur; Jeffrey E Cohen; Jeremy R McGarvey; Yasuhiro Shudo; Jay B Patel; Alen Trubelja; Alexander S Fairman; Bryan B Edwards; George Hung; William Hiesinger; Andrew B Goldstone; Pavan Atluri; Robert L Wilensky; James J Pilla; Joseph H Gorman; Robert C Gorman; Y Joseph Woo
Journal:  Circ Res       Date:  2013-12-23       Impact factor: 17.367

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