Literature DB >> 18023524

In vitro clonal analysis of murine pluripotent stem cells isolated from skeletal muscle and adipose stromal cells.

Jamie Case1, Tamara L Horvath, Christopher B Ballas, Keith L March, Edward F Srour.   

Abstract

OBJECTIVE: Possible clinical utility of pluripotent stem cells (PSCs) with multilineage differentiation capacity depends on their ability to adapt to tissue-specific differentiation conditions. Previous data from our laboratory suggest that putative PSCs exhibiting an immunophenotype of CD45(-)Sca-1+CD117(-)CD90+ can be isolated from multiple tissues. In the present study, the clonal in vitro differentiation potential of two isolates of PSCs was examined.
MATERIALS AND METHODS: Clonal analysis of the differentiation potential of skeletal muscle- (SM) and adipose stromal cell (ASC)-derived PSCs into myogenic, adipogenic, and neurogenic cells was investigated by expanding single PSCs prior to specification under three separate differentiation conditions.
RESULTS: Differentiation of SM- and ASC-derived PSCs into myotubes, adipocytes, and neuronal-like cells was evident in clonal cultures promoting differentiation along these lineages. A total of 2.0%, 1.0%, and 0.33% of SM-derived clones demonstrated unipotent, bipotent, and tripotent differentiation, respectively, into combinations of myocytes, adipocytes, and neuronal cells. As a percentage of SM-derived PSCs, tripotent clones comprised 0.016% of total muscle cells. Similar results were obtained with ASC-derived PSCs, suggesting phenotypic and functional similarities between PSCs from both tissues. Following differentiation of single PSCs into three lineages, a clear and complete commitment to tissue-specific gene expression accompanied by inactivation of lineage-unrelated genes could not be demonstrated in several SM- and ASC-derived clones.
CONCLUSIONS: These data demonstrate that phenotypically defined PSCs remain functionally heterogeneous at the single-cell level and illustrate that morphologic lineage commitment may be independent of exclusive expression and/or loss of associated lineage specific genes.

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Year:  2007        PMID: 18023524      PMCID: PMC2553759          DOI: 10.1016/j.exphem.2007.09.003

Source DB:  PubMed          Journal:  Exp Hematol        ISSN: 0301-472X            Impact factor:   3.084


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