Literature DB >> 18023012

Cognitive reserve hypothesis: Pittsburgh Compound B and fluorodeoxyglucose positron emission tomography in relation to education in mild Alzheimer's disease.

Nina M Kemppainen1, Sargo Aalto, Mira Karrasch, Kjell Någren, Nina Savisto, Vesa Oikonen, Matti Viitanen, Riitta Parkkola, Juha O Rinne.   

Abstract

OBJECTIVE: The reduced risk for Alzheimer's disease (AD) in high-educated individuals has been proposed to reflect brain cognitive reserve, which would provide more efficient compensatory mechanisms against the underlying pathology, and thus delayed clinical expression. Our aim was to find possible differences in brain amyloid ligand 11C-labeled Pittsburgh Compound B ([11C]PIB) uptake and glucose metabolism in high- and low-educated patients with mild AD.
METHODS: Twelve high-educated and 13 low-educated patients with the same degree of cognitive deterioration were studied with PET using [11C]PIB and 18F-fluorodeoxyglucose as ligands. The between-group differences were analyzed with voxel-based statistical method, and quantitative data were obtained with automated region-of-interest analysis.
RESULTS: High-educated patients showed increased [11C]PIB uptake in the lateral frontal cortex compared with low-educated patients. Moreover, high-educated patients had significantly lower glucose metabolic rate in the temporoparietal cortical regions compared with low-educated patients.
INTERPRETATION: Our results suggesting more advanced pathological and functional brain changes in high-educated patients with mild AD are in accordance with the brain cognitive reserve hypothesis and point out the importance of development of reliable markers of underlying AD pathology for early AD diagnostics.

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Year:  2008        PMID: 18023012     DOI: 10.1002/ana.21212

Source DB:  PubMed          Journal:  Ann Neurol        ISSN: 0364-5134            Impact factor:   10.422


  92 in total

Review 1.  Noninvasive brain stimulation in Alzheimer's disease: systematic review and perspectives for the future.

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2.  Amyloid and metabolic positron emission tomography imaging of cognitively normal adults with Alzheimer's parents.

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Review 3.  Amyloid positron emission tomography and cognitive reserve.

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Review 4.  Dissecting Complex and Multifactorial Nature of Alzheimer's Disease Pathogenesis: a Clinical, Genomic, and Systems Biology Perspective.

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5.  Influence of functional connectivity and structural MRI measures on episodic memory.

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6.  A neuroimaging approach to capture cognitive reserve: Application to Alzheimer's disease.

Authors:  Anna C van Loenhoud; Alle Meije Wink; Colin Groot; Sander C J Verfaillie; Jos Twisk; Frederik Barkhof; Bart van Berckel; Philip Scheltens; Wiesje M van der Flier; Rik Ossenkoppele
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7.  Effects of cognitive reserve depend on executive and semantic demands of the task.

Authors:  R Ryan Darby; Michael Brickhouse; David A Wolk; Bradford C Dickerson
Journal:  J Neurol Neurosurg Psychiatry       Date:  2017-06-19       Impact factor: 10.154

8.  Episodic memory loss is related to hippocampal-mediated beta-amyloid deposition in elderly subjects.

Authors:  E C Mormino; J T Kluth; C M Madison; G D Rabinovici; S L Baker; B L Miller; R A Koeppe; C A Mathis; M W Weiner; W J Jagust
Journal:  Brain       Date:  2008-11-28       Impact factor: 13.501

9.  [11C]PIB, [18F]FDG and MR imaging in patients with mild cognitive impairment.

Authors:  A Brück; J R Virta; J Koivunen; J Koikkalainen; N M Scheinin; H Helenius; K Någren; S Helin; R Parkkola; M Viitanen; J O Rinne
Journal:  Eur J Nucl Med Mol Imaging       Date:  2013-06-26       Impact factor: 9.236

Review 10.  Beta-amyloid deposition and the aging brain.

Authors:  Karen M Rodrigue; Kristen M Kennedy; Denise C Park
Journal:  Neuropsychol Rev       Date:  2009-11-12       Impact factor: 7.444

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