Literature DB >> 17621826

A comparison of experimental methods in molecular chronic rhinosinusitis research.

Murugappan Ramanathan1, Andrew P Lane.   

Abstract

BACKGROUND: Research into the molecular pathogenesis of chronic rhinosinusitis (CRS) requires the collection and analysis of sinonasal tissue. Recent gene expression studies have used either surgical tissue specimens or isolated epithelial cell preparations. Here, we compare cultures of nasal epithelial cells, nasal brush biopsy, and whole ethmoid mucosa with respect to expression of innate immune genes.
METHODS: Ethmoid mucosa was collected intraoperatively from 12 CRS and control patients. This tissue either was processed whole for mRNA extraction or was used to generate primary nasal epithelial cell cultures. After 6 weeks, epithelial cells in culture were assessed for multiple innate immune proteins by flow cytometry. In parallel, middle meatal brush biopsy specimens were obtained from the same patients and studied acutely in a similar fashion by flow cytometry. Expression of innate immune genes was determined in whole tissue samples by real-time polymerase chain reaction.
RESULTS: Flow cytometry revealed that brush biopsy specimens contain 75% epithelial cells, whereas primary nasal epithelial cell cultures were pure. Epithelial cells derived from individual subjects expressed very similar levels of innate immune markers whether studied acutely or after 6 weeks in culture. Whole tissue mRNA levels were variable and not correlated to epithelial expression.
CONCLUSION: The choice of experimental methodology can greatly influence the results and interpretation of CRS research. Primary nasal epithelial cells maintain their innate immune receptor expression profile when grown in prolonged culture in vitro. These findings imply that alterations in innate immune gene expression in CRS may be intrinsic to the epithelial cells, even outside of their in vivo microenvironment.

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Year:  2007        PMID: 17621826     DOI: 10.2500/ajr.2007.21.3034

Source DB:  PubMed          Journal:  Am J Rhinol        ISSN: 1050-6586


  23 in total

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Journal:  J Mol Med (Berl)       Date:  2014-11-13       Impact factor: 4.599

2.  Bactericidal antibiotics promote reactive oxygen species formation and inflammation in human sinonasal epithelial cells.

Authors:  Michael A Kohanski; Anuj Tharakan; Andrew P Lane; Murugappan Ramanathan
Journal:  Int Forum Allergy Rhinol       Date:  2015-12-01       Impact factor: 3.858

3.  Treatment-recalcitrant chronic rhinosinusitis with polyps is associated with altered epithelial cell expression of interleukin-33.

Authors:  Douglas D Reh; Yadong Wang; Murugappan Ramanathan; Andrew P Lane
Journal:  Am J Rhinol Allergy       Date:  2010 Mar-Apr       Impact factor: 2.467

4.  Chitin stimulates expression of acidic mammalian chitinase and eotaxin-3 by human sinonasal epithelial cells in vitro.

Authors:  Ashley Lalaker; Louis Nkrumah; Won-Kyung Lee; Murugappan Ramanathan; Andrew P Lane
Journal:  Am J Rhinol Allergy       Date:  2009 Jan-Feb       Impact factor: 2.467

5.  Fungal extracts stimulate solitary chemosensory cell expansion in noninvasive fungal rhinosinusitis.

Authors:  Neil N Patel; Vasiliki Triantafillou; Ivy W Maina; Alan D Workman; Charles C L Tong; Edward C Kuan; Peter Papagiannopoulos; John V Bosso; Nithin D Adappa; James N Palmer; Michael A Kohanski; De'Broski R Herbert; Noam A Cohen
Journal:  Int Forum Allergy Rhinol       Date:  2019-03-20       Impact factor: 3.858

6.  Nasal polyp fibroblasts modulate epithelial characteristics via Wnt signaling.

Authors:  Alex Dobzanski; Syed Muaz Khalil; Andrew P Lane
Journal:  Int Forum Allergy Rhinol       Date:  2018-08-17       Impact factor: 3.858

7.  Reversal of cigarette smoke extract-induced sinonasal epithelial cell barrier dysfunction through Nrf2 Activation.

Authors:  Anuj Tharakan; Ashleigh A Halderman; Andrew P Lane; Shyam Biswal; Murugappan Ramanathan
Journal:  Int Forum Allergy Rhinol       Date:  2016-08-31       Impact factor: 3.858

8.  Mouse nasal epithelial innate immune responses to Pseudomonas aeruginosa quorum-sensing molecules require taste signaling components.

Authors:  Robert J Lee; Bei Chen; Kevin M Redding; Robert F Margolskee; Noam A Cohen
Journal:  Innate Immun       Date:  2013-09-17       Impact factor: 2.680

9.  Damage-associated molecular patterns stimulate interleukin-33 expression in nasal polyp epithelial cells.

Authors:  Gina Paris; Tatyana Pozharskaya; Tomefa Asempa; Andrew P Lane
Journal:  Int Forum Allergy Rhinol       Date:  2014-01       Impact factor: 3.858

Review 10.  A role for airway taste receptor modulation in the treatment of upper respiratory infections.

Authors:  Jennifer E Douglas; Cecil J Saunders; Danielle R Reed; Noam A Cohen
Journal:  Expert Rev Respir Med       Date:  2016-01-22       Impact factor: 3.772

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