Literature DB >> 17204702

In vivo evaluation of P-glycoprotein modulation of 8 PET radioligands used clinically.

Kiichi Ishiwata1, Kazunori Kawamura, Kazuhiko Yanai, N Harry Hendrikse.   

Abstract

UNLABELLED: P-glycoprotein (P-gp) regulates the ability of endogenous and exogenous compounds to cross the blood-brain barrier. We investigated whether PET tracers used clinically for studying brain function are affected by P-gp.
METHODS: Modulation of 8 radioligands by P-gp was assayed in mice by evaluating the effect of treatment with cyclosporine A (CsA) on uptake into the brain (assay 1) and the effect of treatment with a cold ligand of the corresponding radioligand on uptake of (11)C-verapamil, a representative radioligand for P-gp (assay 2). Brain-to-blood ratios were also examined as the other index to correct the delivery of radioligands. The radioligands investigated were (11)C-TMSX (adenosine A(2A) receptor), (11)C-MPDX (adenosine A(1) receptor), (11)C-PK11195 (peripheral benzodiazepine receptor), (11)C-flumazenil (central benzodiazepine receptor), (11)C-raclopride (dopamine D(2)-like receptor), (11)C-pyrilamine (histamine H(1) receptor), (11)C-PIB (amyloid plaque), and (11)C-donepezil (acetylcholine esterase).
RESULTS: In assay 1, CsA treatment increased both the uptake and the brain-to-blood ratio of (11)C-TMSX, (11)C-MPDX, (11)C-flumazenil, and (11)C-donepezil among the 8 radioligands. In assay 2, in which 4 cold ligands were examined, cold verapamil slightly increased the brain-to-blood ratio of (11)C-verapamil, but TMSX, MPDX, and MPPF did not increase either parameter.
CONCLUSION: Assay 1 was suitable for evaluating the P-gp modulation of radioligands. Among the 8 radioligands investigated, (11)C-TMSX, (11)C-MPDX, (11)C-flumazenil, and (11)C-donepezil were modulated by P-gp.

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Year:  2007        PMID: 17204702

Source DB:  PubMed          Journal:  J Nucl Med        ISSN: 0161-5505            Impact factor:   10.057


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