Literature DB >> 16901770

Assessment of fibrosis progression in untreated irish women with chronic hepatitis C contracted from immunoglobulin anti-D.

Robert A Levine1, Schuyler O Sanderson, Robert Ploutz-Snyder, Frank Murray, Elaine Kay, John Hegarty, Niamh Nolan, Dermot Kelleher, George McDonald, J Conor O'Keane, John Crowe.   

Abstract

BACKGROUND & AIMS: In 1996 we initiated a retrospective-prospective study in 184 untreated women infected in 1977 with chronic hepatitis C virus (HCV). To provide insight into the natural history of HCV, we determined liver fibrosis outcomes and any predictors of such.
METHODS: Baseline 1994 biopsy specimens (size, >or=15 mm; portal areas, >or=5) and sequential biopsy specimens were assessed by Ishak score for grade change (increase or decrease of >or=2 points) and stage progression or regression (increase or reduction of >or=1 point), the latter correlated with digital quantification of fibrosis percentage.
RESULTS: No baseline biopsy specimens had cirrhosis, therefore all could potentially progress. Grade and stage scores decreased or increased significantly in 28% and 18% and 24% and 27% of patients, respectively. There was a positive correlation between baseline and sequential grade/stage scores (r = .39, P < .001), and between semiquantitative Ishak scores and fibrosis percentage (Spearman rho = .85; P < .01). Baseline alanine transaminase values (mean, 49 U/L; range, 23-363 U/L) correlated positively with changes in grade (r = .41, P < .01) and stage (r = .39, P < .01), and regression analyses indicated that baseline alanine transaminase value was a good predictor of such changes. Confounding variables (alcohol, smoking, and herbal and paracetamol [acetaminophen] use) did not correlate with histologic outcomes.
CONCLUSIONS: In a follow-up study, 49% of patients showed no change in fibrosis, 24% showed regression, and only 27% showed progression, including 4 patients (2.1%) who developed stage 6 cirrhosis. Unidirectional sequential grade/stage concordance attested to biopsy sample reliability. Given the current age of these women in their fifth decade, some still may have a risk for more advanced liver disease, but for most of these patients it appears unlikely.

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Year:  2006        PMID: 16901770     DOI: 10.1016/j.cgh.2006.05.028

Source DB:  PubMed          Journal:  Clin Gastroenterol Hepatol        ISSN: 1542-3565            Impact factor:   11.382


  15 in total

1.  Digital quantification is more precise than traditional semiquantitation of hepatic steatosis: correlation with fibrosis in 220 treatment-naïve patients with chronic hepatitis C.

Authors:  Sekou R Rawlins; Ola El-Zammar; J Michael Zinkievich; Nancy Newman; Robert A Levine
Journal:  Dig Dis Sci       Date:  2010-05-12       Impact factor: 3.199

2.  Elevated serum CK18 levels in chronic hepatitis C patients are associated with advanced fibrosis but not steatosis.

Authors:  A B Jazwinski; A J Thompson; P J Clark; S Naggie; H L Tillmann; K Patel
Journal:  J Viral Hepat       Date:  2011-11-24       Impact factor: 3.728

3.  Liver disease in women: the influence of gender on epidemiology, natural history, and patient outcomes.

Authors:  Jennifer Guy; Marion G Peters
Journal:  Gastroenterol Hepatol (N Y)       Date:  2013-10

Review 4.  Systematic review: identifying patients with chronic hepatitis C in need of early treatment and intensive monitoring--predictors and predictive models of disease progression.

Authors:  M A Konerman; S Yapali; A S Lok
Journal:  Aliment Pharmacol Ther       Date:  2014-08-28       Impact factor: 8.171

Review 5.  KASL clinical practice guidelines: management of hepatitis C.

Authors: 
Journal:  Clin Mol Hepatol       Date:  2016-03-28

6.  Poor response to pegylated interferon and ribavirin in older women infected with hepatitis C virus of genotype 1b in high viral loads.

Authors:  Hitomi Sezaki; Fumitaka Suzuki; Yusuke Kawamura; Hiromi Yatsuji; Tetsuya Hosaka; Norio Akuta; Masahiro Kobayashi; Yoshiyuki Suzuki; Satoshi Saitoh; Yasuji Arase; Kenji Ikeda; Yuzo Miyakawa; Hiromitsu Kumada
Journal:  Dig Dis Sci       Date:  2008-10-28       Impact factor: 3.199

7.  CD4+ T-lymphocyte telomere length is related to fibrosis stage, clinical outcome and treatment response in chronic hepatitis C virus infection.

Authors:  Matthew Hoare; William T H Gelson; Abhi Das; Jean M Fletcher; Susan E Davies; Martin D Curran; Sarah L Vowler; Mala K Maini; Arne N Akbar; Graeme J M Alexander
Journal:  J Hepatol       Date:  2010-04-22       Impact factor: 25.083

8.  Fibrosis progression in African Americans and Caucasian Americans with chronic hepatitis C.

Authors:  Norah A Terrault; Kelly Im; Ross Boylan; Peter Bacchetti; David E Kleiner; Robert J Fontana; Jay H Hoofnagle; Steven H Belle
Journal:  Clin Gastroenterol Hepatol       Date:  2008-08-19       Impact factor: 11.382

Review 9.  The history of the "natural history" of hepatitis C (1968-2009).

Authors:  Leonard B Seeff
Journal:  Liver Int       Date:  2009-01       Impact factor: 5.828

10.  Histological changes in HCV antibody-positive, HCV RNA-negative subjects suggest persistent virus infection.

Authors:  Matthew Hoare; William T H Gelson; Simon M Rushbrook; Martin D Curran; Tracy Woodall; Nicholas Coleman; Susan E Davies; Graeme J M Alexander
Journal:  Hepatology       Date:  2008-12       Impact factor: 17.425

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