Literature DB >> 16820589

Endothelial Protection, AT1 blockade and Cholesterol-Dependent Oxidative Stress: the EPAS trial.

Henning Morawietz1, Sandra Erbs, Jürgen Holtz, Andreas Schubert, Michael Krekler, Winfried Goettsch, Oliver Kuss, Volker Adams, Karsten Lenk, Friedrich W Mohr, Gerhard Schuler, Rainer Hambrecht.   

Abstract

BACKGROUND: Statins and angiotensin type 1 (AT1) receptor blockers reduce cardiovascular mortality and morbidity. In the Endothelial Protection, AT1 blockade and Cholesterol-Dependent Oxidative Stress (EPAS) trial, impact of independent or combined statin and AT1 receptor blocker therapy on endothelial expression of anti-atherosclerotic and proatherosclerotic genes and endothelial function in arteries of patients with coronary artery disease were tested. METHODS AND
RESULTS: Sixty patients with stable coronary artery disease undergoing elective coronary artery bypass grafting (CABG) surgery were randomized 4 weeks before surgery to: (A) control without inhibition of renin-angiotensin system or statin; (B) statin (pravastatin 40 mg/d); (C) AT1 blockade (irbesartan 150 mg/d); or (D) combination of statin and AT1 blocker in same dosages. Primary end point was a priori therapy-dependent regulation of an anti-atherosclerotic endothelial expression quotient Q including mRNA expression (in arbitrary units measured by real-time polymerase chain reaction) of endothelial nitric oxide synthase and C-type natriuretic peptide, divided by expression of oxidized low-density lipoprotein receptor LOX-1 and NAD(P)H oxidase subunit gp91phox in left internal mammary arteries biopsies obtained by CABG surgery; 49 patients completed the study. Statin therapy increased lnQ from 3.2+/-0.4 to 4.4+/-0.4 significantly versus control. AT(1) blockade showed a trend to increase lnQ to 4.2+/-0.5. Combination of statin and AT1 blocker further increased lnQ to 5.1+/-0.6, but a putative interaction of both therapies in lnQ was not significant. Furthermore, preoperative therapy with statin, AT1 blocker and their combination improved endothelial function in internal mammary artery rings.
CONCLUSIONS: Statin and AT1 blocker therapy independently and in combination improve an anti-atherosclerotic endothelial expression quotient and endothelial function.

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Year:  2006        PMID: 16820589     DOI: 10.1161/CIRCULATIONAHA.105.001313

Source DB:  PubMed          Journal:  Circulation        ISSN: 0009-7322            Impact factor:   29.690


  21 in total

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Review 3.  Role of lipotoxicity in endothelial dysfunction.

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Review 4.  Comparing angiotensin II receptor blockers on benefits beyond blood pressure.

Authors:  Helmy M Siragy
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5.  Association of scavenger receptors in adipose tissue with insulin resistance in nondiabetic humans.

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6.  Techniques to harvest diseased human peripheral arteries and measure endothelial function in an ex vivo model.

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7.  Angiotensin II Blockade and Total Cardiovascular Risk : Beyond Blood Pressure Reduction.

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Journal:  High Blood Press Cardiovasc Prev       Date:  2013-01-22

8.  The HMG-CoA reductase inhibitor rosuvastatin and the angiotensin receptor antagonist candesartan attenuate atherosclerosis in an apolipoprotein E-deficient mouse model of diabetes via effects on advanced glycation, oxidative stress and inflammation.

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9.  Augmentation of the renin-angiotensin system by hypercholesterolemia promotes vascular diseases.

Authors:  Alan Daugherty; Hong Lu; Debra L Rateri; Lisa A Cassis
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Review 10.  Statin prophylaxis and inflammatory mediators following cardiopulmonary bypass: a systematic review.

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