Literature DB >> 16630954

Associations of cord blood fatty acids with lymphocyte proliferation, IL-13, and IFN-gamma.

Diane R Gold1, Ben M Willwerth, Kelan G Tantisira, Patricia W Finn, Bianca Schaub, David L Perkins, Arthur Tzianabos, Ngoc P Ly, Christian Schroeter, Fiona Gibbons, Hannia Campos, Emily Oken, Matthew W Gillman, Lyle J Palmer, Louise M Ryan, Scott T Weiss.   

Abstract

BACKGROUND: N-3 and n-6 polyunsaturated fatty acids (PUFAs) have been hypothesized to have opposing influences on neonatal immune responses that might influence the risk of allergy or asthma. However, both n-3 eicosapentaenoic acid (EPA) and n-6 arachidonic acid (AA) are required for normal fetal development.
OBJECTIVE: We evaluated whether cord blood fatty acid levels were related to neonatal immune responses and whether n-3 and n-6 PUFA responses differed.
METHODS: We examined the relation of cord blood plasma n-3 and n-6 PUFAs (n = 192) to antigen- and mitogen-stimulated cord blood lymphocyte proliferation (n = 191) and cytokine (IL-13 and IFN-gamma; n = 167) secretion in a US birth cohort.
RESULTS: Higher levels of n-6 linoleic acid were correlated with higher IL-13 levels in response to Bla g 2 (cockroach, P = .009) and Der f 1 (dust mite, P = .02). Higher n-3 EPA and n-6 AA levels were each correlated with reduced lymphocyte proliferation and IFN-gamma levels in response to Bla g 2 and Der f 1 stimulation. Controlling for potential confounders, EPA and AA had similar independent effects on reduced allergen-stimulated IFN-gamma levels. If neonates had either EPA or AA levels in the highest quartile, their Der f 1 IFN-gamma levels were 90% lower (P = .0001) than those with both EPA and AA levels in the lowest 3 quartiles. Reduced AA/EPA ratio was associated with reduced allergen-stimulated IFN-gamma level.
CONCLUSION: Increased levels of fetal n-3 EPA and n-6 AA might have similar effects on attenuation of cord blood lymphocyte proliferation and IFN-gamma secretion. CLINICAL IMPLICATIONS: The implications of these findings for allergy or asthma development are not yet known.

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Year:  2006        PMID: 16630954      PMCID: PMC1508138          DOI: 10.1016/j.jaci.2005.12.1322

Source DB:  PubMed          Journal:  J Allergy Clin Immunol        ISSN: 0091-6749            Impact factor:   10.793


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