Literature DB >> 16313289

Effect of GB virus C on response to antiretroviral therapy in HIV-infected Brazilians.

I E Souza1, W Zhang, R S Diaz, K Chaloner, D Klinzman, J T Stapleton.   

Abstract

OBJECTIVES: GB virus C (GBV-C) infection is associated with delayed mortality in HIV-infected people in most, but not all, studies. Previous investigations of the effect of GBV-C viraemia on response to antiretroviral therapy (ART) were inconclusive. To determine the effect of GBV-C on ART, we retrospectively analysed plasma samples taken from patients in a prospective randomized clinical trial of ART in HIV-positive Brazilians.
METHODS: GBV-C viraemia was characterized by testing stored serum samples from 175 participants by reverse transcriptase-polymerase chain reaction (RT-PCR). Subjects were randomized to receive indinavir (n=59), zidovudine and lamivudine (n=58), or zidovudine, lamivudine and indinavir (n=58). The effect of GBV-C viraemia on the average change in HIV viral load and CD4 count following initiation of therapy was evaluated in a multiple regression analysis.
RESULTS: The prevalence of GBV-C viraemia was similar to that observed in previous studies (24%). HIV viral load decreased following ART to a significantly greater extent in patients with GBV-C viraemia (by 0.48 log(10) HIV-1 RNA copies/mL, P=0.009, adjusting for age, ART group, and baseline CD4 count). Although there was no significant difference in change in CD4 count between individuals with and without GBV-C viraemia overall, CD4 counts were higher following 48 weeks of therapy in GBV-C viraemic individuals receiving the least potent ART regimen (zidovudine and lamivudine) compared with those without GBV-C infection.
CONCLUSIONS: GBV-C viraemia is associated with an enhanced reduction of HIV viral load in response to ART. In this study of treatment-naive individuals during 48 weeks of follow up, patients with GBV-C viraemia had reductions in HIV viral load that were approximately 0.5 log copies/mL greater than those found in patients without GBV-C viraemia. This is similar to reductions observed with nucleoside reverse transcriptase inhibitors.

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Year:  2006        PMID: 16313289     DOI: 10.1111/j.1468-1293.2005.00339.x

Source DB:  PubMed          Journal:  HIV Med        ISSN: 1464-2662            Impact factor:   3.180


  11 in total

1.  Pace of Coreceptor Tropism Switch in HIV-1-Infected Individuals after Recent Infection.

Authors:  Muhammad Shoaib Arif; James Hunter; Ana Rachel Léda; Jean Paulo Lopes Zukurov; Sadia Samer; Michelle Camargo; Juliana Galinskas; Esper Georges Kallás; Shirley Vasconcelos Komninakis; Luiz Mario Janini; Maria Cecilia Sucupira; Ricardo Sobhie Diaz
Journal:  J Virol       Date:  2017-09-12       Impact factor: 5.103

2.  Effect of primer selection on estimates of GB virus C (GBV-C) prevalence and response to antiretroviral therapy for optimal testing for GBV-C viremia.

Authors:  I E Souza; J B Allen; J Xiang; D Klinzman; R Diaz; S Zhang; K Chaloner; D Zdunek; G Hess; C F Williams; L Benning; J T Stapleton
Journal:  J Clin Microbiol       Date:  2006-09       Impact factor: 5.948

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9.  Faster HIV-1 disease progression among Brazilian individuals recently infected with CXCR4-utilizing strains.

Authors:  Maria Cecilia Araripe Sucupira; Sabri Sanabani; Rodrigo M Cortes; Maria Teresa M Giret; Helena Tomiyama; Mariana M Sauer; Ester Cerdeira Sabino; Luiz Mario Janini; Esper Georges Kallas; Ricardo Sobhie Diaz
Journal:  PLoS One       Date:  2012-01-26       Impact factor: 3.240

10.  Exploring Viral Interference Using Peptides: Molecular Determinants of HIV-1 Inhibition by a Peptide Derived from Human Pegivirus-1 Envelope Protein E2.

Authors:  Rebecca Hoffmann; Tamara Ruegamer; Johanna Schaubächer; Anette Rohrhofer; Peter Kirmeß; Karen M Fiebig; Barbara Schmidt; Jutta Eichler
Journal:  ChemMedChem       Date:  2021-02-03       Impact factor: 3.466

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