THE OBJECTIVE: The purpose of this research was to establish if determined at the reception level of troponin I could be such kind of factor. Presented data represent preliminary report of this study. MATERIAL AND METHODS: The analyzed data base on 196 patients with IS: 166 discharged from hospital and 30 with clinical course terminated by death. The IS was confirmed by computed tomography (CT). The conducted recording consists of data like e.g.: age, sex and risk stroke factors. Also the following factors were evaluated on admission: state of consciousness, level of neurological deficiency according to Scandinavian Stroke Scale (SSS). After half of year again SSS level was evaluated. RESULTS: The studied groups differed in level of neurological deficiency determined on admission. The direct mortality within IS group and increased level of troponin I represents 20.90% and with correct level of troponin I represents 12.40% (p=NS). The half year mortality group recording has not been considered yet because research is still continuing and database is continually updated. CONCLUSION: The increased level of troponin I on admission is not a prognostic factor of direct mortality within IS patients. We noticed that occurrence of disturbances in patient's EKG places those patients in the group of increased risk of death.
THE OBJECTIVE: The purpose of this research was to establish if determined at the reception level of troponin I could be such kind of factor. Presented data represent preliminary report of this study. MATERIAL AND METHODS: The analyzed data base on 196 patients with IS: 166 discharged from hospital and 30 with clinical course terminated by death. The IS was confirmed by computed tomography (CT). The conducted recording consists of data like e.g.: age, sex and risk stroke factors. Also the following factors were evaluated on admission: state of consciousness, level of neurological deficiency according to Scandinavian Stroke Scale (SSS). After half of year again SSS level was evaluated. RESULTS: The studied groups differed in level of neurological deficiency determined on admission. The direct mortality within IS group and increased level of troponin I represents 20.90% and with correct level of troponin I represents 12.40% (p=NS). The half year mortality group recording has not been considered yet because research is still continuing and database is continually updated. CONCLUSION: The increased level of troponin I on admission is not a prognostic factor of direct mortality within IS patients. We noticed that occurrence of disturbances in patient's EKG places those patients in the group of increased risk of death.