Literature DB >> 16215305

Intermittent androgen castration: a biological reality during intermittent treatment in metastatic prostate cancer?

N Mottet1, C Lucas, E Sene, C Avances, L Maubach, J M Wolff.   

Abstract

INTRODUCTION: To assess the effects of intermittent maximal androgen blockade (IMAB) on testosterone (T) levels during on- and off-treatment periods.
MATERIALS AND METHODS: A total of 51 patients with metastatic prostate cancer underwent a 6-months period of continuous maximal androgen blockade (MAB) consisting of leuprorelin (3.75 mg at monthly intervals) plus flutamide (250 mg t.i.d.) followed by IMAB. During each cycle, the cut-off prostate-specific antigen (PSA) levels to stop and resume treatment were 4 and 10 ng/ml, respectively. IMAB continued until progression under treatment occurred. Monthly PSA and T measurements were performed in central laboratories.
RESULTS: From the 51 patients included (mean age 67.6 years), 27, 16, 12, 8 and 5 underwent a second, third, fourth, fifth and sixth cycle, respectively (mean follow up: 17 months). Before treatment, 4 patients had a T lower than normal laboratory value but these recovered all to a normal T value at the end of the first cycle. During the 6 cycles, only 8 patients did not recover a normal T at least once during the off-treatment periods (OTP). The mean T values at the end of each OTP did not change during these 6 cycles (Anova test, p=0.621) with a mean stable recovery delay of 32-43 days (Anova test, p=0.722).
CONCLUSION: IMAB protocol with an initial 6-month treatment period can result in an intermittent castration with the recovery of normal T levels in most patients during six consecutive cycles of treatment. Copyright (c) 2005 S. Karger AG, Basel.

Entities:  

Mesh:

Substances:

Year:  2005        PMID: 16215305     DOI: 10.1159/000087794

Source DB:  PubMed          Journal:  Urol Int        ISSN: 0042-1138            Impact factor:   2.089


  3 in total

1.  Prolongation of off-cycle interval by finasteride is not associated with survival improvement in intermittent androgen deprivation therapy in LNCaP tumor model.

Authors:  Yujuan Wang; Shubham Gupta; Vi Hua; Raquel Ramos-Garcia; Daniel Shevrin; Borko D Jovanovic; Joel B Nelson; Zhou Wang
Journal:  Prostate       Date:  2010-02-01       Impact factor: 4.104

Review 2.  Androgen deprivation therapy for the treatment of prostate cancer: consider both benefits and risks.

Authors:  Hendrik Isbarn; Laurent Boccon-Gibod; Peter R Carroll; Francesco Montorsi; Claude Schulman; Matthew R Smith; Cora N Sternberg; Urs E Studer
Journal:  Eur Urol       Date:  2008-10-14       Impact factor: 20.096

Review 3.  DNA licensing as a novel androgen receptor mediated therapeutic target for prostate cancer.

Authors:  Jason M D'Antonio; Donald J Vander Griend; John T Isaacs
Journal:  Endocr Relat Cancer       Date:  2009-02-24       Impact factor: 5.678

  3 in total

北京卡尤迪生物科技股份有限公司 © 2022-2023.