Literature DB >> 15870125

The relationships of adiponectin with insulin and lipids are strengthened with increasing adiposity.

Lisa J Martin1, Jessica G Woo, Stephen R Daniels, Elizabeth Goodman, Lawrence M Dolan.   

Abstract

CONTEXT: Adipose tissue inflammation has been implicated in the pathogenesis of obesity-related comorbidities. Adiponectin, an antiinflammatory protein, improves insulin sensitivity and lipid levels systemically. Because adiponectin is secreted by adipocytes, it may also act locally to counteract insulin resistance and dyslipidemia worsened by inflammation.
OBJECTIVE: The aim of this study was to determine whether associations between adiponectin and insulin sensitivity and lipids are stronger with increasing adiposity.
DESIGN: This cross-sectional study involved participants in The Princeton School District Study.
SETTING: The study was conducted in the Princeton City schools (Cincinnati, OH) during the 2001-2002 school year. PARTICIPANTS: A total of 1196 non-Hispanic White and Black students in grades 5-12 participated. MAIN OUTCOME MEASURE: The relationships between adiponectin and high-density lipoprotein, triglycerides, and insulin were measured. To test our hypothesis, we: 1) compared correlation and regression coefficients of lean and nonlean individuals, and 2) incorporated an adiponectin by adiposity interaction in regression models.
RESULTS: For high-density lipoprotein and triglycerides, the relationship with adiponectin, although present among lean adolescents, strengthened with increasing adiposity. However, with insulin, a relationship with adiponectin was only present among nonlean adolescents.
CONCLUSIONS: These analyses suggest that adiponectin's relationship with insulin and lipids strengthens with increasing adiposity, such that heavier adolescents have a greater benefit from high levels of adiponectin than their lean counterparts.

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Year:  2005        PMID: 15870125      PMCID: PMC4313924          DOI: 10.1210/jc.2005-0019

Source DB:  PubMed          Journal:  J Clin Endocrinol Metab        ISSN: 0021-972X            Impact factor:   5.958


  27 in total

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Review 2.  The inflammatory syndrome: the role of adipose tissue cytokines in metabolic disorders linked to obesity.

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3.  Adiponectin, a new member of the family of soluble defense collagens, negatively regulates the growth of myelomonocytic progenitors and the functions of macrophages.

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4.  Correlation of the adipocyte-derived protein adiponectin with insulin resistance index and serum high-density lipoprotein-cholesterol, independent of body mass index, in the Japanese population.

Authors:  Yukihiro Yamamoto; Hiroshi Hirose; Ikuo Saito; Motowo Tomita; Matsuo Taniyama; Koichi Matsubara; Yasunori Okazaki; Tatsuya Ishii; Kanako Nishikai; Takao Saruta
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5.  Decreased plasma adiponectin concentrations in women with dyslipidemia.

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9.  Adiponectin differentially regulates cytokines in porcine macrophages.

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10.  Diet-induced insulin resistance in mice lacking adiponectin/ACRP30.

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  24 in total

1.  Paradoxically high adiponectin and the healthy obese phenotype in obese black and white 16-year-old girls.

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2.  Increased arterial stiffness is found in adolescents with obesity or obesity-related type 2 diabetes mellitus.

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4.  Paradoxically high adiponectin in obese 16-year-old girls protects against appearance of the metabolic syndrome and its components seven years later.

Authors:  John A Morrison; Charles J Glueck; Stephen Daniels; Ping Wang; Davis Stroop
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5.  Underdiagnosis of Metabolic Syndrome in Non-Hispanic Black Adolescents: A Call for Ethnic-Specific Criteria.

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6.  Serum adiponectin complexes and cardiovascular risk in children with chronic kidney disease.

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7.  Serum adiponectin in relation to race-ethnicity and vascular risk factors in the Northern Manhattan Study.

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9.  Resting IL-6 and TNF-α level in children of different weight and fitness status.

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10.  Gender differences in the relationships among obesity, adiponectin and brachial artery distensibility in adolescents and young adults.

Authors:  E M Urbina; P Khoury; L J Martin; D D'Alessio; L M Dolan
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