Literature DB >> 15732031

Relationship between oxidative stress and systolic dysfunction in patients with hypertrophic cardiomyopathy.

Kazufumi Nakamura1, Kengo Fukushima Kusano, Hiromi Matsubara, Yoichi Nakamura, Aya Miura, Nobuhiro Nishii, Kimikazu Banba, Satoshi Nagase, Katsumasa Miyaji, Hiroshi Morita, Hironori Saito, Tetsuro Emori, Tohru Ohe.   

Abstract

BACKGROUND: Progression of hypertrophic cardiomyopathy (HCM) to left ventricular dilatation and systolic dysfunction sometimes occurs. However, the mechanism is not known. We examined whether oxidative stress was elevated in myocardia of HCM patients and whether the levels were correlated with left ventricular dilatation and systolic dysfunction. METHODS AND
RESULTS: Endomyocardial biopsy samples obtained from the right ventricular side of the septum of 31 patients with HCM, and 10 control subjects were studied immunohistochemically for the expression of 4-hydroxy-2-nonenal (HNE)-modified protein, which is a major lipid peroxidation product. Expression of HNE-modified protein was found in all myocardial biopsy samples from patients with HCM. Expression was distinct in the cytosol of cardiomyocytes. The expression levels in patients with HCM were significantly increased compared with those in control subjects (P = .0005). The expression levels in patients with HCM were correlated with left ventricular end-diastolic diameter (r = 0.483, P = .0053) and end-systolic diameter (r = 0.500, P = .0037) determined by echocardiography. The expression levels were inversely correlated with left ventricular ejection fraction determined by left ventriculography (r = -0.640, P = .0001).
CONCLUSION: Oxidative stress was elevated in myocardia of HCM patients and the levels were correlated with left ventricular dilatation and systolic dysfunction. Oxidative stress is involved in the pathogenesis of heart failure in patients with HCM.

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Year:  2005        PMID: 15732031     DOI: 10.1016/j.cardfail.2004.05.005

Source DB:  PubMed          Journal:  J Card Fail        ISSN: 1071-9164            Impact factor:   5.712


  25 in total

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Review 2.  Uncoupling proteins in heart failure.

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Review 3.  Targeting aldehyde dehydrogenase 2: new therapeutic opportunities.

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4.  Hypertrophic Cardiomyopathy: A Vicious Cycle Triggered by Sarcomere Mutations and Secondary Disease Hits.

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Journal:  Antioxid Redox Signal       Date:  2018-04-11       Impact factor: 8.401

5.  Allele-specific differences in transcriptome, miRNome, and mitochondrial function in two hypertrophic cardiomyopathy mouse models.

Authors:  Styliani Vakrou; Ryuya Fukunaga; D Brian Foster; Lars Sorensen; Yamin Liu; Yufan Guan; Kirubel Woldemichael; Roberto Pineda-Reyes; Ting Liu; Jill C Tardiff; Leslie A Leinwand; Carlo G Tocchetti; Theodore P Abraham; Brian O'Rourke; Miguel A Aon; M Roselle Abraham
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6.  Aldehydic load and aldehyde dehydrogenase 2 profile during the progression of post-myocardial infarction cardiomyopathy: benefits of Alda-1.

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7.  Aldehyde dehydrogenase 2 activation in heart failure restores mitochondrial function and improves ventricular function and remodelling.

Authors:  Katia M S Gomes; Juliane C Campos; Luiz R G Bechara; Bruno Queliconi; Vanessa M Lima; Marie-Helene Disatnik; Paulo Magno; Che-Hong Chen; Patricia C Brum; Alicia J Kowaltowski; Daria Mochly-Rosen; Julio C B Ferreira
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9.  Comparison of biomarkers of oxidative stress and cardiovascular disease in humans and chimpanzees (Pan troglodytes).

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10.  Importance of the bioenergetic reserve capacity in response to cardiomyocyte stress induced by 4-hydroxynonenal.

Authors:  Bradford G Hill; Brian P Dranka; Luyun Zou; John C Chatham; Victor M Darley-Usmar
Journal:  Biochem J       Date:  2009-10-23       Impact factor: 3.857

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