Literature DB >> 15699840

Prone position delays the progression of ventilator-induced lung injury in rats: does lung strain distribution play a role?

Franco Valenza1, Massimiliano Guglielmi, Micol Maffioletti, Cecilia Tedesco, Patrizia Maccagni, Tommaso Fossali, Gabriele Aletti, Giuliana Anna Porro, Manuela Irace, Eleonora Carlesso, Nadia Carboni, Marco Lazzerini, Luciano Gattinoni.   

Abstract

OBJECTIVE: To investigate if prone position delays the progression of experimental ventilator-induced lung injury, possibly due to a more homogeneous distribution of strain within lung parenchyma.
DESIGN: Prospective, randomized, controlled trial.
SETTING: Animal laboratory of a university hospital.
SUBJECTS: Thirty-five Sprague Dawley male rats (weight 257 +/- 45 g).
INTERVENTIONS: Mechanical ventilation in either supine or prone position and computed tomography scan analysis. MEASUREMENTS: : Animals were ventilated in supine (n = 15) or prone (n = 15) position until a similar ventilator-induced lung injury was reached. To do so, experiments were interrupted when respiratory system elastance was 150% of baseline. Ventilator-induced lung injury was assessed as lung wet-to-dry ratio and histology. Time to reach lung injury was considered as a main outcome measure. In five additional animals, computed tomography scans (GE Light Speed QX/I, thickness 1.25 mm, interval 0.6 mm, 100 MA, 100 Kv) were randomly taken at end-expiration and end-inspiration in both positions, and quantitative analysis was performed. Data are shown as mean +/- sd.
MEASUREMENTS AND MAIN RESULTS: Similar ventilator-induced lung injury was reached (respiratory system elastance, wet-to-dry ratio, and histology). The time taken to achieve the target ventilator-induced lung injury was longer with prone position (73 +/- 37 mins vs. 112 +/- 42, supine vs. prone, p = .011). Computed tomography scan analysis performed before lung injury revealed that at end-expiration, the lung was wider in prone position (p = .004) and somewhat shorter (p = .09), despite similar lung volumes (p = .455). Lung density along the vertical axis increased significantly only in supine position (p = .002). Lung strain was greater in supine as opposed to prone position (width strain, 7.8 +/- 1.8% vs. 5.6 +/- 0.9, supine vs. prone, p = .029).
CONCLUSIONS: Prone position delays the progression of ventilator-induced lung injury. Computed tomography scan analysis suggests that a more homogeneous distribution of strain may be implicated in the protective role of prone position against ventilator-induced lung injury.

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Year:  2005        PMID: 15699840     DOI: 10.1097/01.ccm.0000150660.45376.7c

Source DB:  PubMed          Journal:  Crit Care Med        ISSN: 0090-3493            Impact factor:   7.598


  43 in total

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5.  The Effects of Prone Position Ventilation on Experimental Mild Acute Lung Injury Induced by Intraperitoneal Lipopolysaccharide Injection in Rats.

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7.  Ventilation in the prone position: for some but not for all?

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Review 8.  Mechanisms of the effects of prone positioning in acute respiratory distress syndrome.

Authors:  C Guerin; L Baboi; J C Richard
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10.  Relationship between gas exchange response to prone position and lung recruitability during acute respiratory failure.

Authors:  Alessandro Protti; Davide Chiumello; Massimo Cressoni; Eleonora Carlesso; Cristina Mietto; Virna Berto; Marco Lazzerini; Michael Quintel; Luciano Gattinoni
Journal:  Intensive Care Med       Date:  2009-02-03       Impact factor: 17.440

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