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Literature DB >> 15650756

BI-1 protects cells from oxygen glucose deprivation by reducing the calcium content of the endoplasmic reticulum.

B C Westphalen, J Wessig, F Leypoldt, S Arnold, A Methner.

Abstract

Entities: Chemical Disease

Mesh：

Substances：

Year: 2005 PMID： 15650756 DOI： 10.1038/sj.cdd.4401547

Source DB: PubMed Journal: Cell Death Differ ISSN： 1350-9047 Impact factor: 15.828

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Cited

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18 in total

1. Endoplasmic reticulum protein BI-1 modulates unfolded protein response signaling and protects against stroke and traumatic brain injury.

Authors: Maryla Krajewska; Lucy Xu; Wenjie Xu; Stan Krajewski; Christina L Kress; Jiankun Cui; Li Yang; Fumitoshi Irie; Yu Yamaguchi; Stuart A Lipton; John C Reed
Journal: Brain Res Date: 2010-11-12 Impact factor: 3.252

2. BAX inhibitor-1 is a Ca(2+) channel critically important for immune cell function and survival.

Authors: D Lisak; T Schacht; A Gawlitza; P Albrecht; O Aktas; B Koop; M Gliem; H H Hofstetter; K Zanger; G Bultynck; J B Parys; H De Smedt; T Kindler; P Adams-Quack; M Hahn; A Waisman; J C Reed; N Hövelmeyer; A Methner
Journal: Cell Death Differ Date: 2015-10-16 Impact factor: 15.828

Review 3. TMBIM-mediated Ca²⁺ homeostasis and cell death.

Authors: Qun Liu
Journal: Biochim Biophys Acta Mol Cell Res Date: 2017-01-05 Impact factor: 4.739

4. Plant Bax Inhibitor-1 interacts with ATG6 to regulate autophagy and programmed cell death.

Authors: Guoyong Xu; Shanshan Wang; Shaojie Han; Ke Xie; Yan Wang; Jinlin Li; Yule Liu
Journal: Autophagy Date: 2017-07-03 Impact factor: 16.016

5. BAX inhibitor-1 regulates autophagy by controlling the IRE1α branch of the unfolded protein response.

Authors: Karen Castillo; Diego Rojas-Rivera; Fernanda Lisbona; Benjamín Caballero; Melissa Nassif; Felipe A Court; Sebastian Schuck; Consuelo Ibar; Peter Walter; Jimena Sierralta; Alvaro Glavic; Claudio Hetz
Journal: EMBO J Date: 2011-09-16 Impact factor: 11.598

6. The C terminus of Bax inhibitor-1 forms a Ca2+-permeable channel pore.

Authors: Geert Bultynck; Santeri Kiviluoto; Nadine Henke; Hristina Ivanova; Lars Schneider; Volodymyr Rybalchenko; Tomas Luyten; Koen Nuyts; Wim De Borggraeve; Ilya Bezprozvanny; Jan B Parys; Humbert De Smedt; Ludwig Missiaen; Axel Methner
Journal: J Biol Chem Date: 2011-11-28 Impact factor: 5.157

Review 10. Bax Inhibitor-1, a conserved cell death suppressor, is a key molecular switch downstream from a variety of biotic and abiotic stress signals in plants.

Authors: Naohide Watanabe; Eric Lam
Journal: Int J Mol Sci Date: 2009-07-10 Impact factor: 6.208

BI-1 protects cells from oxygen glucose deprivation by reducing the calcium content of the endoplasmic reticulum.

1. Endoplasmic reticulum protein BI-1 modulates unfolded protein response signaling and protects against stroke and traumatic brain injury.

2. BAX inhibitor-1 is a Ca(2+) channel critically important for immune cell function and survival.

Review 3. TMBIM-mediated Ca²⁺ homeostasis and cell death.

4. Plant Bax Inhibitor-1 interacts with ATG6 to regulate autophagy and programmed cell death.

5. BAX inhibitor-1 regulates autophagy by controlling the IRE1α branch of the unfolded protein response.

6. The C terminus of Bax inhibitor-1 forms a Ca2+-permeable channel pore.

7. Structural basis for a pH-sensitive calcium leak across membranes.

8. BI-1 regulates endoplasmic reticulum Ca2+ homeostasis downstream of Bcl-2 family proteins.

Review 9. The selective BH4-domain biology of Bcl-2-family members: IP3Rs and beyond.

Review 10. Bax Inhibitor-1, a conserved cell death suppressor, is a key molecular switch downstream from a variety of biotic and abiotic stress signals in plants.

Review 3. TMBIM-mediated Ca2+ homeostasis and cell death.

Review 9. The selective BH4-domain biology of Bcl-2-family members: IP3Rs and beyond.

Review 10. Bax Inhibitor-1, a conserved cell death suppressor, is a key molecular switch downstream from a variety of biotic and abiotic stress signals in plants.

Review 3. TMBIM-mediated Ca²⁺ homeostasis and cell death.