Literature DB >> 15534189

A novel polymerase gamma mutation in a family with ophthalmoplegia, neuropathy, and Parkinsonism.

Michelangelo Mancuso1, Massimiliano Filosto, Shin J Oh, Salvatore DiMauro.   

Abstract

BACKGROUND: Mutations in polymerase gamma cause progressive external ophthalmoplegia and a variety of associated symptoms and signs, including neuropathy, ataxia, hypogonadism, hearing loss, muscle weakness, and psychiatric problems. Extrapyramidal signs have been rarely described.
OBJECTIVE: To describe a family with a novel polymerase gamma mutation and autosomal dominant transmission of progressive external ophthalmoplegia, neuropathy, hypogonadism, and parkinsonism.
DESIGN: Case report. PATIENTS: The proband, a 49-year-old woman with incipient parkinsonism, and her 59-year-old brother with overt parkinsonian features. MAIN OUTCOME MEASURES: Mutation in the proband by sequencing the polymerase gamma gene and in affected relatives by restriction fragment length polymorphism analysis.
RESULTS: We found multiple mitochondrial DNA deletions in the proband's muscle and a novel missense mutation in the polymerase gamma gene (A2492G) in the proband and in her affected siblings.
CONCLUSION: Parkinsonism was a prominent clinical feature in this family with autosomal dominant ophthalmoplegia, multiple mitochondrial DNA deletions, and a novel mutation in the polymerase gamma gene.

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Year:  2004        PMID: 15534189     DOI: 10.1001/archneur.61.11.1777

Source DB:  PubMed          Journal:  Arch Neurol        ISSN: 0003-9942


  25 in total

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7.  Genetic analysis of two Japanese families with progressive external ophthalmoplegia and parkinsonism.

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9.  Molecular and clinical genetics of mitochondrial diseases due to POLG mutations.

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Review 10.  Role of the mitochondrial DNA replication machinery in mitochondrial DNA mutagenesis, aging and age-related diseases.

Authors:  Karen L DeBalsi; Kirsten E Hoff; William C Copeland
Journal:  Ageing Res Rev       Date:  2016-04-30       Impact factor: 10.895

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