Literature DB >> 15372234

A novel activation-induced cytidine deaminase gene mutation in a Tunisian family with hyper IgM syndrome.

Claudia Fiorini1, Sawssen Jilani, Claretta Gioia Losi, Antonietta Silini, Silvia Giliani, Simona Ferrari, Luigi D Notarangelo, Alessandro Plebani, Taher Sfar, Ahmed Helal.   

Abstract

UNLABELLED: Mutations in activation-induced cytidine deaminase can cause an autosomal recessive form of hyper-IgM syndrome. We have examined a Tunisian family composed of six members: two healthy parents, their two healthy daughters and two affected sons. We found a homozygous transversion G to T in the two sons while heterozygosity for the mutation was found in all other family members. This alteration is localised in intron 2 at the +1 position resulting in defective splicing. Use of various intronic cryptic splice-sites led to expression of various aberrant mRNA species.
CONCLUSION: This is a novel mutation found in the gene encoding for activation-induced cytidine deaminase in a Tunisian family with hyper-IgM type 2 syndrome. This alteration leads to the use of two cryptic splicing sites causing the formation of two different mRNA species.

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Year:  2004        PMID: 15372234     DOI: 10.1007/s00431-004-1540-8

Source DB:  PubMed          Journal:  Eur J Pediatr        ISSN: 0340-6199            Impact factor:   3.183


  11 in total

1.  Specific missense mutations in NEMO result in hyper-IgM syndrome with hypohydrotic ectodermal dysplasia.

Authors:  A Jain; C A Ma; S Liu; M Brown; J Cohen; W Strober
Journal:  Nat Immunol       Date:  2001-03       Impact factor: 25.606

2.  Mutations in activation-induced cytidine deaminase in patients with hyper IgM syndrome.

Authors:  Y Minegishi; A Lavoie; C Cunningham-Rundles; P M Bédard; J Hébert; L Côté; K Dan; D Sedlak; R H Buckley; A Fischer; A Durandy; M E Conley
Journal:  Clin Immunol       Date:  2000-12       Impact factor: 3.969

3.  Mutations of CD40 gene cause an autosomal recessive form of immunodeficiency with hyper IgM.

Authors:  S Ferrari; S Giliani; A Insalaco; A Al-Ghonaium; A R Soresina; M Loubser; M A Avanzini; M Marconi; R Badolato; A G Ugazio; Y Levy; N Catalan; A Durandy; A Tbakhi; L D Notarangelo; A Plebani
Journal:  Proc Natl Acad Sci U S A       Date:  2001-10-23       Impact factor: 11.205

4.  Specific expression of activation-induced cytidine deaminase (AID), a novel member of the RNA-editing deaminase family in germinal center B cells.

Authors:  M Muramatsu; V S Sankaranand; S Anant; M Sugai; K Kinoshita; N O Davidson; T Honjo
Journal:  J Biol Chem       Date:  1999-06-25       Impact factor: 5.157

5.  Type two hyper-IgM syndrome caused by mutation in activation-induced cytidine deaminase.

Authors:  Yi Zhu; Shigeaki Nonoyama; Tomohiro Morio; Masamichi Muramatsu; Tasuku Honjo; Shuki Mizutani
Journal:  J Med Dent Sci       Date:  2003-03

Review 6.  Cross-talk between CD40 and CD40L: lessons from primary immune deficiencies.

Authors:  Simona Ferrari; Alessandro Plebani
Journal:  Curr Opin Allergy Clin Immunol       Date:  2002-12

7.  Clinical, immunologic and genetic analysis of 29 patients with autosomal recessive hyper-IgM syndrome due to Activation-Induced Cytidine Deaminase deficiency.

Authors:  Pierre Quartier; Jacinta Bustamante; Ozden Sanal; Alessandro Plebani; Marianne Debré; Anne Deville; Jiri Litzman; Jacov Levy; Jean-Paul Fermand; Peter Lane; Gerd Horneff; Guzide Aksu; Isik Yalçin; Graham Davies; Ilhan Tezcan; Furgen Ersoy; Nadia Catalan; Kohsuhe Imai; Alain Fischer; Anne Durandy
Journal:  Clin Immunol       Date:  2004-01       Impact factor: 3.969

8.  CD40 ligand gene defects responsible for X-linked hyper-IgM syndrome.

Authors:  R C Allen; R J Armitage; M E Conley; H Rosenblatt; N A Jenkins; N G Copeland; M A Bedell; S Edelhoff; C M Disteche; D K Simoneaux
Journal:  Science       Date:  1993-02-12       Impact factor: 47.728

9.  Human uracil-DNA glycosylase deficiency associated with profoundly impaired immunoglobulin class-switch recombination.

Authors:  Kohsuke Imai; Geir Slupphaug; Wen-I Lee; Patrick Revy; Shigeaki Nonoyama; Nadia Catalan; Leman Yel; Monique Forveille; Bodil Kavli; Hans E Krokan; Hans D Ochs; Alain Fischer; Anne Durandy
Journal:  Nat Immunol       Date:  2003-09-07       Impact factor: 25.606

10.  Activation-induced cytidine deaminase (AID) deficiency causes the autosomal recessive form of the Hyper-IgM syndrome (HIGM2).

Authors:  P Revy; T Muto; Y Levy; F Geissmann; A Plebani; O Sanal; N Catalan; M Forveille; R Dufourcq-Labelouse; A Gennery; I Tezcan; F Ersoy; H Kayserili; A G Ugazio; N Brousse; M Muramatsu; L D Notarangelo; K Kinoshita; T Honjo; A Fischer; A Durandy
Journal:  Cell       Date:  2000-09-01       Impact factor: 41.582

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  3 in total

Review 1.  Evaluation of molecular models for the affinity maturation of antibodies: roles of cytosine deamination by AID and DNA repair.

Authors:  Mala Samaranayake; Janusz M Bujnicki; Michael Carpenter; Ashok S Bhagwat
Journal:  Chem Rev       Date:  2006-02       Impact factor: 60.622

2.  Novel and recurrent AID mutations underlie prevalent autosomal recessive form of HIGM in consanguineous patients.

Authors:  Hanen Ouadani; Imen Ben-Mustapha; Meriem Ben-ali; Leila Ben-khemis; Beya Larguèche; Raoudha Boussoffara; Sonia Maalej; Ilhem Fetni; Saida Hassayoun; Abdelmajid Mahfoudh; Fethi Mellouli; Sadok Yalaoui; Hatem Masmoudi; Mohamed Bejaoui; Mohamed-Ridha Barbouche
Journal:  Immunogenetics       Date:  2015-11-06       Impact factor: 2.846

Review 3.  Lessons from Genetic Studies of Primary Immunodeficiencies in a Highly Consanguineous Population.

Authors:  Mohamed-Ridha Barbouche; Najla Mekki; Meriem Ben-Ali; Imen Ben-Mustapha
Journal:  Front Immunol       Date:  2017-06-27       Impact factor: 7.561

  3 in total

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