Literature DB >> 15362033

Analysis of hepatitis C virus quasispecies transmission and evolution in patients infected through blood transfusion.

Tomasz Laskus1, Jeffrey Wilkinson, Juan F Gallegos-Orozco, Marek Radkowski, Debra M Adair, Marek Nowicki, Eva Operskalski, Zelma Buskell, Leonard B Seeff, Hugo Vargas, Jorge Rakela.   

Abstract

BACKGROUND & AIMS: Studies on hepatitis C virus (HCV) quasispecies dynamics in the natural course of infection are rare owing to difficulties in obtaining samples from the early phase of infection.
METHODS: We studied 15 patients from the Transfusion-Transmitted Viruses Study who seroconverted to anti-HCV after receiving infected blood. Follow-up serum samples were collected every 2-3 weeks for 6 months, at 10 months, and at 11-16 years. Viral quasispecies in the second envelope hypervariable region 1 (E2/HVR1) and 5' untranslated region (5'UTR) were analyzed with single-strand conformation polymorphism (SSCP) and heteroduplex mobility assay (HMA).
RESULTS: Seven patients cleared infection within 7-24 weeks (mean, 14.0 wk) and 3 patients eventually became anti-HCV negative. In 6 patients with resolving hepatitis the SSCP band pattern remained stable, whereas in one patient minor changes appeared before clearance. In contrast, in all 8 patients progressing to chronicity, major changes in the E2/HVR1 quasispecies developed at 8-22 weeks (mean, 13.1 wk). Shannon entropy and medium mobility shift values derived from HMA gels remained stable in patients with resolving hepatitis but changed in those who developed chronic infection. Only 2 patients showed minor changes in 5'UTR. A decrease in E2/HVR1 complexity at the time of transmission (bottleneck) was found in 5 patients altogether.
CONCLUSIONS: Changes in E2/HVR1 quasispecies 8-22 weeks after infection, likely caused by mounting immune pressure, were predictive of ensuing chronic infection, whereas stability was associated with resolution. Our study also showed that composition of HCV quasispecies may be preserved during transmission from host to host.

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Year:  2004        PMID: 15362033     DOI: 10.1053/j.gastro.2004.06.005

Source DB:  PubMed          Journal:  Gastroenterology        ISSN: 0016-5085            Impact factor:   22.682


  27 in total

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2.  Functional characterization of core genes from patients with acute hepatitis C virus infection.

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4.  Presence of hepatitis C virus (HCV) RNA in the genital tracts of HCV/HIV-1-coinfected women.

Authors:  Marek J Nowicki; Tomasz Laskus; Georgia Nikolopoulou; Marek Radkowski; Jeffrey Wilkinson; Wenbo B Du; Jorge Rakela; Andrea Kovacs
Journal:  J Infect Dis       Date:  2005-09-29       Impact factor: 5.226

5.  Establishment of a novel permissive cell line for the propagation of hepatitis C virus by expression of microRNA miR122.

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7.  Wide range of quasispecies diversity during primary hepatitis C virus infection.

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Journal:  J Virol       Date:  2005-04       Impact factor: 5.103

8.  Influence of quasispecies on virological responses and disease severity in patients with chronic hepatitis C.

Authors:  Deepak Kumar; Abdul Malik; Mohammad Asim; Anita Chakravarti; Rakha-H Das; Premashis Kar
Journal:  World J Gastroenterol       Date:  2008-02-07       Impact factor: 5.742

9.  Three different functional microdomains in the hepatitis C virus hypervariable region 1 (HVR1) mediate entry and immune evasion.

Authors:  Mo Guan; Wenbo Wang; Xiaoqing Liu; Yimin Tong; Yuan Liu; Hao Ren; Shiying Zhu; Jean Dubuisson; Thomas F Baumert; Yongzhe Zhu; Haoran Peng; Laure Aurelian; Ping Zhao; Zhongtian Qi
Journal:  J Biol Chem       Date:  2012-08-27       Impact factor: 5.157

10.  Evidence of recombination in Hepatitis C Virus populations infecting a hemophiliac patient.

Authors:  Pilar Moreno; Macarena Alvarez; Lilia López; Gonzalo Moratorio; Didier Casane; Matías Castells; Silvia Castro; Juan Cristina; Rodney Colina
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