Literature DB >> 15248047

Neonatal oral administration of DiaPep277, combined with hydrolysed casein diet, protects against Type 1 diabetes in BB-DP rats. An experimental study.

S Brugman1, F A Klatter2, J Visser3, N A Bos2, D Elias4, J Rozing2.   

Abstract

AIMS/HYPOTHESIS: Environmental factors such as diet and bacterial antigens play an important role in the onset of Type 1 diabetes. Different self-antigens are suggested to play a role in the development of diabetes. Antibodies against the 60-kDa heat shock protein 60, which have a high homology to bacterial heat shock protein 65, have been found in the circulation at the onset of diabetes in humans and in pre-diabetic NOD-mice. One of the immunodominant epitopes in autoimmune diabetes is p277, a specific peptide of human heat shock protein 60 corresponding to positions 437-460. In this study we investigated whether neonatal oral administration of DiaPep277 (a synthetic peptide analogue of p277) affected the development of diabetes in the BioBreeding-Diabetes Prone (BB-DP) rat, and whether this could potentiate the effect of a protective hydrolysed casein-diet.
METHODS: BB-DP rats were orally inoculated once per day with placebo or DiaPep277 at days 4, 5, 6 and 7 of life. At the age of 21 days rats were weaned on to a conventional, cereal-based diet or on to the hydrolysed casein-diet.
RESULTS: The development of diabetes in animals receiving DiaPep277 in combination with the hydrolysed casein-diet was delayed by 17 days, and a relative reduction of the incidence by 64% was seen. Non-diabetic animals did not show any sign of insulitis. CONCLUSIONS/
INTERPRETATION: Short-term neonatal feeding with p277 in early life, combined with diet adaptation, appears to provide a procedure to significantly reduce the development of Type 1 diabetes in later life.

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Year:  2004        PMID: 15248047     DOI: 10.1007/s00125-004-1452-1

Source DB:  PubMed          Journal:  Diabetologia        ISSN: 0012-186X            Impact factor:   10.122


  7 in total

1.  Hsp60 peptide therapy of NOD mouse diabetes induces a Th2 cytokine burst and downregulates autoimmunity to various beta-cell antigens.

Authors:  D Elias; A Meilin; V Ablamunits; O S Birk; P Carmi; S Könen-Waisman; I R Cohen
Journal:  Diabetes       Date:  1997-05       Impact factor: 9.461

Review 2.  Cellular and molecular pathogenic mechanisms of insulin-dependent diabetes mellitus.

Authors:  J W Yoon; H S Jun
Journal:  Ann N Y Acad Sci       Date:  2001-04       Impact factor: 5.691

3.  Oral administration of insulin to neonates suppresses spontaneous and cyclophosphamide induced diabetes in the NOD mouse.

Authors:  R Maron; M Guerau-de-Arellano; X Zhang; H L Weiner
Journal:  J Autoimmun       Date:  2001-02       Impact factor: 7.094

Review 4.  Peptide therapy for Type I diabetes: the immunological homunculus and the rationale for vaccination.

Authors:  I R Cohen
Journal:  Diabetologia       Date:  2002-09-05       Impact factor: 10.122

5.  Potential mechanisms by which certain foods promote or inhibit the development of spontaneous diabetes in BB rats: dose, timing, early effect on islet area, and switch in infiltrate from Th1 to Th2 cells.

Authors:  F W Scott; H E Cloutier; R Kleemann; U Wöerz-Pagenstert; P Rowsell; H W Modler; H Kolb
Journal:  Diabetes       Date:  1997-04       Impact factor: 9.461

6.  Short-term dietary adjustment with a hydrolyzed casein-based diet postpones diabetes development in the diabetes-prone BB rat.

Authors:  Jeroen Visser; Sylvia Brugman; Flip Klatter; Lotte Vis; Herman Groen; Jan Strubbe; Jan Rozing
Journal:  Metabolism       Date:  2003-03       Impact factor: 8.694

7.  Neonatal tolerization with glutamic acid decarboxylase but not with bovine serum albumin delays the onset of diabetes in NOD mice.

Authors:  J S Petersen; A E Karlsen; H Markholst; A Worsaae; T Dyrberg; B Michelsen
Journal:  Diabetes       Date:  1994-12       Impact factor: 9.461

  7 in total
  8 in total

1.  Antibiotic treatment partially protects against type 1 diabetes in the Bio-Breeding diabetes-prone rat. Is the gut flora involved in the development of type 1 diabetes?

Authors:  S Brugman; F A Klatter; J T J Visser; A C M Wildeboer-Veloo; H J M Harmsen; J Rozing; N A Bos
Journal:  Diabetologia       Date:  2006-07-01       Impact factor: 10.122

Review 2.  Type 1 diabetes and gut microbiota: Friend or foe?

Authors:  Changyun Hu; F Susan Wong; Li Wen
Journal:  Pharmacol Res       Date:  2015-03-05       Impact factor: 7.658

3.  Restoration of impaired intestinal barrier function by the hydrolysed casein diet contributes to the prevention of type 1 diabetes in the diabetes-prone BioBreeding rat.

Authors:  J T J Visser; K Lammers; A Hoogendijk; M W Boer; S Brugman; S Beijer-Liefers; A Zandvoort; H Harmsen; G Welling; F Stellaard; N A Bos; A Fasano; J Rozing
Journal:  Diabetologia       Date:  2010-09-19       Impact factor: 10.122

Review 4.  Tight junctions, intestinal permeability, and autoimmunity: celiac disease and type 1 diabetes paradigms.

Authors:  Jeroen Visser; Jan Rozing; Anna Sapone; Karen Lammers; Alessio Fasano
Journal:  Ann N Y Acad Sci       Date:  2009-05       Impact factor: 5.691

5.  Heat-shock protein gene is not associated with type-1 diabetes in African Americans.

Authors:  Noureddine Berka; Gail N Bland; El Hajja Erabhaoui; Georgia M Dunston
Journal:  J Natl Med Assoc       Date:  2007-07       Impact factor: 1.798

6.  Early life treatment with vancomycin propagates Akkermansia muciniphila and reduces diabetes incidence in the NOD mouse.

Authors:  C H F Hansen; L Krych; D S Nielsen; F K Vogensen; L H Hansen; S J Sørensen; K Buschard; A K Hansen
Journal:  Diabetologia       Date:  2012-05-10       Impact factor: 10.122

Review 7.  The Role of Heat Shock Proteins in Type 1 Diabetes.

Authors:  Abu Saleh Md Moin; Manjula Nandakumar; Abdoulaye Diane; Mohammed Dehbi; Alexandra E Butler
Journal:  Front Immunol       Date:  2021-01-14       Impact factor: 7.561

8.  Prevention of diabetes by a hydrolysed casein-based diet in diabetes-prone BioBreeding rats does not involve restoration of the defective natural regulatory T cell function.

Authors:  J Visser; J L Hillebrands; M Walther Boer; N A Bos; J Rozing
Journal:  Diabetologia       Date:  2009-04-25       Impact factor: 10.122

  8 in total

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