Literature DB >> 15203054

Abnormalities of prepulse inhibition do not depend on blink reflex excitability: a study in Parkinson's disease and Huntington's disease.

J Valls-Solé1, J E Muñoz, F Valldeoriola.   

Abstract

OBJECTIVE: Prepulse inhibition of the blink reflex is a robust phenomenon with an interesting physiology and a large potential for clinical applicability. In the study presented here we investigated whether the blink reflex inhibition by a prepulse (BRIP) is influenced by the blink reflex excitability recovery (BRER).
METHODS: The study was undertaken in 20 patients with Parkinson's disease (PD), 20 patients with Huntington's disease (HD) and 20 healthy volunteers. BRER was determined by measuring the size of the response to a test supraorbital nerve stimulus as a percentage of the response to a conditioning stimulus at inter-stimuli intervals of 100-1000 ms. BRIP was determined as the percentage reduction induced in the response to a supraorbital nerve stimulus by either a low intensity auditory click or a weak third finger somatosensory stimulus, applied with a leading interval of 50-110 ms.
RESULTS: There was a negative correlation between the percentage BRER and the percentage BRIP (Pearson's correlation coefficient of -0.37). BRER was enhanced in 14 PD patients (70%) and 6 HD patients (30%), while it was depressed in 10 HD patients (50%). BRIP was significantly reduced in 15 PD patients (75%) and 16 HD patients (80%). No significant correlation was found between abnormally enhanced BRER and abnormally reduced BRIP in all patients as a group (chi(2)=2.4;P=0.11). A weak correlation was found in PD patients (P=0.019) and no correlation was observed in HD patients (P=0.8).
CONCLUSIONS: Our results indicate that an abnormally reduced BRIP was not always accompanied by an abnormally enhanced BRER in patients with HD. The two tests likely assess specific and distinct brainstem functions, and provide different types of information. While BRIP may be the result of a widespread integrative processing of sensory stimuli, BRER likely reflects the excitability of a chain of brainstem inter-neurons. SIGNIFICANCE: BRER and BRIP provide independent information on the state of functionally separate circuits that converge on trigemino-facial brainstem inter-neurons.

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Year:  2004        PMID: 15203054     DOI: 10.1016/j.clinph.2004.02.014

Source DB:  PubMed          Journal:  Clin Neurophysiol        ISSN: 1388-2457            Impact factor:   3.708


  28 in total

1.  Trigeminal nerve stimulation modulates brainstem more than cortical excitability in healthy humans.

Authors:  B Mercante; G Pilurzi; F Ginatempo; A Manca; P Follesa; E Tolu; F Deriu
Journal:  Exp Brain Res       Date:  2015-08-11       Impact factor: 1.972

2.  Reduced startle gating after D1 blockade: effects of concurrent D2 blockade.

Authors:  Neal R Swerdlow; Jody M Shoemaker; Michele J Bongiovanni; Alaina C Neary; Laura S Tochen; Richard L Saint Marie
Journal:  Pharmacol Biochem Behav       Date:  2005-09-26       Impact factor: 3.533

Review 3.  Update: studies of prepulse inhibition of startle, with particular relevance to the pathophysiology or treatment of Tourette Syndrome.

Authors:  Neal R Swerdlow
Journal:  Neurosci Biobehav Rev       Date:  2012-09-24       Impact factor: 8.989

4.  Neurophysiological biomarkers informing the clinical neuroscience of schizophrenia: mismatch negativity and prepulse inhibition of startle.

Authors:  Gregory A Light; Neal R Swerdlow
Journal:  Curr Top Behav Neurosci       Date:  2014

5.  Sensorimotor gating deficits in schizophrenia: Advancing our understanding of the phenotype, its neural circuitry and genetic substrates.

Authors:  Neal R Swerdlow; Gregory A Light
Journal:  Schizophr Res       Date:  2018-03-07       Impact factor: 4.939

6.  Reorganization of sensory input at brainstem in hemifacial spasm and postparalytic facial syndrome.

Authors:  Meral E Kızıltan; Ayşegul Gunduz
Journal:  Neurol Sci       Date:  2017-11-09       Impact factor: 3.307

Review 7.  Sensorimotor gating deficits in "two-hit" models of schizophrenia risk factors.

Authors:  Asma Khan; Susan B Powell
Journal:  Schizophr Res       Date:  2017-10-22       Impact factor: 4.939

8.  Jendrassik maneuver effect on spinal and brainstem reflexes.

Authors:  Lale Aslihan Ertuglu; Asli Aydin; Hatice Kumru; Josep Valls-Sole; Eloy Opisso; Serpil Cecen; Kemal S Türker
Journal:  Exp Brain Res       Date:  2019-10-24       Impact factor: 1.972

9.  Separable noradrenergic and dopaminergic regulation of prepulse inhibition in rats: implications for predictive validity and Tourette Syndrome.

Authors:  Neal R Swerdlow; Michele J Bongiovanni; Laura Tochen; Jody M Shoemaker
Journal:  Psychopharmacology (Berl)       Date:  2006-04-01       Impact factor: 4.530

Review 10.  Realistic expectations of prepulse inhibition in translational models for schizophrenia research.

Authors:  Neal R Swerdlow; Martin Weber; Ying Qu; Gregory A Light; David L Braff
Journal:  Psychopharmacology (Berl)       Date:  2008-06-21       Impact factor: 4.530

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