Literature DB >> 15007085

Azacitidine induces demethylation of the Epstein-Barr virus genome in tumors.

Anthony T C Chan1, Qian Tao, Keith D Robertson, Ian W Flinn, Risa B Mann, Barbara Klencke, Wing Hong Kwan, Thomas Wai-Tong Leung, Philip J Johnson, Richard F Ambinder.   

Abstract

PURPOSE: To determine whether therapy with a DNA methyltransferase inhibitor is effective in achieving demethylation and gene re-expression in tumor DNA in patients.
METHODS: Biopsy specimens were obtained from patients with Epstein-Barr virus-associated tumors, enrolled on a clinical trial of 5-azacitidine, within 72 hours of the conclusion of the last infusion of the first cycle of therapy, and compared to pretreatment specimens. Methylation-specific polymerase chain reaction, bisulfite genomic sequencing, and immunohistochemistry were used to assess demethylation and gene re-expression.
RESULTS: Substantial degrees of demethylation were detected in all latent and lytic Epstein-Barr virus promoters examined. Immunohistochemistry suggested activation of a previously silent viral antigen expression in one instance.
CONCLUSION: Pharmacologic reversal of dense CpG methylation in tumor tissue can be achieved in patients.

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Year:  2004        PMID: 15007085     DOI: 10.1200/JCO.2004.04.185

Source DB:  PubMed          Journal:  J Clin Oncol        ISSN: 0732-183X            Impact factor:   44.544


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