Literature DB >> 14966089

EBV-positive gastric adenocarcinomas: a distinct clinicopathologic entity with a low frequency of lymph node involvement.

Josine van Beek1, Axel zur Hausen, Elma Klein Kranenbarg, Cornelis J H van de Velde, Jaap M Middeldorp, Adriaan J C van den Brule, Chris J L M Meijer, Elisabeth Bloemena.   

Abstract

PURPOSE: Epstein-Barr virus (EBV) is detected in a substantial subgroup of gastric adenocarcinomas worldwide. We have previously reported that these EBV-positive gastric carcinomas carry distinct genomic aberrations. In the present study, we analyzed a large cohort of EBV-positive and EBV-negative gastric adenocarcinomas for their clinicopathologic features to determine whether they constitute a different clinical entity. PATIENTS AND METHODS: Using a validated polymerase chain reaction/enzyme immunoassay-based prescreening method in combination with EBER1/2-RNA in situ hybridization, EBV was detected in the tumor cells of 7.2% (n = 41) of the gastric carcinomas from the Dutch D1D2 trial (N = 566; mean follow-up, 9 years). EBV status was correlated with clinicopathologic features collected for the Dutch D1D2 trial.
RESULTS: EBV-positive gastric carcinomas occurred significantly more frequently in males (P <.0001) and in younger patients (P =.012). Most were of the intestinal type according to the Laurén classification (P =.047) or tubular according to the WHO classification (P =.006) and located in the proximal part of the stomach (P <.0001). A significantly lower tumor-node-metastasis system-stage (P =.026) was observed in the patients with EBV-carrying carcinomas, which was solely explained by less lymph node (LN) involvement (P =.034) in these cases. In addition, a better prognosis, as reflected by a longer disease-free period (P =.04) and a significant better cancer-related survival (P =.02), was observed for these patients, which could be explained by less LN involvement, less residual disease, and younger patient age.
CONCLUSION: EBV-carrying gastric adenocarcinomas are a distinct entity of carcinomas, characterized not only by unique genomic aberrations, but also by distinct clinicopathologic features associated with significantly better prognosis.

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Year:  2004        PMID: 14966089     DOI: 10.1200/JCO.2004.08.061

Source DB:  PubMed          Journal:  J Clin Oncol        ISSN: 0732-183X            Impact factor:   44.544


  103 in total

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Review 5.  Epstein Barr virus-associated tumours: an update for the attention of the working pathologist.

Authors:  H-J Delecluse; R Feederle; B O'Sullivan; P Taniere
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Authors:  Jiwon Koh; Keun-Wook Lee; Soo Kyung Nam; An Na Seo; Ji-Won Kim; Jin Won Kim; Do Joong Park; Hyung-Ho Kim; Woo Ho Kim; Hye Seung Lee
Journal:  Oncologist       Date:  2019-08-01

Review 7.  Recapitulating Human Gastric Cancer Pathogenesis: Experimental Models of Gastric Cancer.

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8.  Improved survival of gastric cancer with tumour Epstein-Barr virus positivity: an international pooled analysis.

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Journal:  Gut       Date:  2013-04-12       Impact factor: 23.059

9.  Helicobacter pylori and EBV in gastric carcinomas: methylation status and microsatellite instability.

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Journal:  World J Gastroenterol       Date:  2010-01-21       Impact factor: 5.742

10.  Meta-analysis shows that prevalence of Epstein-Barr virus-positive gastric cancer differs based on sex and anatomic location.

Authors:  Gwen Murphy; Ruth Pfeiffer; M Constanza Camargo; Charles S Rabkin
Journal:  Gastroenterology       Date:  2009-05-13       Impact factor: 22.682

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