Literature DB >> 14739422

Crossed cerebellar diaschisis in patients with cortical infarction: logistic regression analysis to control for confounding effects.

Yuichi Komaba1, Masahiro Mishina, Kouichi Utsumi, Yasuo Katayama, Shiro Kobayashi, Osamu Mori.   

Abstract

BACKGROUND AND
PURPOSE: Crossed cerebellar diaschisis (CCD) refers to reduced metabolism and blood flow in the cerebellar hemisphere contralateral to a cerebral lesion. Many cortical areas have been reported to cause CCD without consideration of confounding factors. We performed single-photon emission computed tomography (SPECT) in patients with cortical infarction to identify regions independently related to CCD, controlling for possible confounding effects.
METHODS: Patients with unilateral cortical infarction (n=113; 75 male, 38 female; mean+/-SD age, 66+/-13 years) underwent SPECT of the brain with N-isopropyl-p-[(123)I]iodoamphetamine ((123)I-IMP). Regional cerebral blood flow was measured autoradiographically. Asymmetry indices (AIs) were calculated on the basis of ratios representing symmetrical regional cerebral blood flow in the cerebellum and 16 cerebral regions. CCD was defined as AI for cerebellum >0.1. AIs for 16 cortical regions were considered for both dichotomous and continuous variables for analysis of CCD occurrence by means of backward logistic regression.
RESULTS: For dichotomized variables, hypoperfusion of postcentral (odds ratio [OR]=7.607; 95% CI, 2.299 to 25.174) and supramarginal (OR=3.916; 95% CI, 1.394 to 11.003) regions independently influenced CCD. For continuous variables, hypoperfusion of postcentral (OR=1.044; 95% CI, 1.019 to 1.068) and supramarginal (OR=1.021; 95% CI, 1.001 to 1.041) regions (and, as a negative factor, medial occipital regions; OR=0.942; 95% CI, 0.895 to 0.991) independently influenced CCD.
CONCLUSIONS: Many cortical areas apparently do not contribute to CCD. Correspondence of CCD between dichotomized and continuous analyses suggests that location of a lesion, not severity, is the main determinant of CCD.

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Year:  2004        PMID: 14739422     DOI: 10.1161/01.STR.0000109771.56160.F5

Source DB:  PubMed          Journal:  Stroke        ISSN: 0039-2499            Impact factor:   7.914


  34 in total

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Journal:  J Neurol       Date:  2010-05-15       Impact factor: 4.849

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Authors:  Wolfgang G Kunz; Wieland H Sommer; Christopher Höhne; Matthias P Fabritius; Felix Schuler; Franziska Dorn; Ahmed E Othman; Felix G Meinel; Louisa von Baumgarten; Maximilian F Reiser; Birgit Ertl-Wagner; Kolja M Thierfelder
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3.  Ultrastructure of Diaschisis Lesions after Traumatic Brain Injury.

Authors:  Clayton A Wiley; Stephanie J Bissel; Andrew Lesniak; C Edward Dixon; Jonathan Franks; Donna Beer Stolz; Ming Sun; Guoji Wang; Robert Switzer; Patrick M Kochanek; Geoffrey Murdoch
Journal:  J Neurotrauma       Date:  2016-03-30       Impact factor: 5.269

4.  Ataxic hemiparesis from strategic frontal white matter infarction with crossed cerebellar diaschisis.

Authors:  Alexander C Flint; MaryAlice C Naley; Clinton B Wright
Journal:  Stroke       Date:  2005-11-23       Impact factor: 7.914

5.  Crossed cerebellar diaschisis as an indicator of severe cerebral hyperperfusion after direct bypass for moyamoya disease.

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8.  Crossed cerebellar diaschisis in status epilepticus.

Authors:  Edgar A Samaniego; Erika Stuckert; Nancy Fischbein; Christine A C Wijman
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9.  Remote effects in the ipsilateral thalamus and/or contralateral cerebellar hemisphere using FDG PET in patients with brain tumors.

Authors:  Hitomi Iwasa; Yoriko Murata; Miki Nishimori; Kana Miyatake; Michiko Tadokoro; Shino Kohsaki; Munenobu Nogami; Yusuke Ueba; Tetsuya Ueba; Takuji Yamagami
Journal:  Jpn J Radiol       Date:  2018-01-25       Impact factor: 2.374

10.  Significance of postoperative crossed cerebellar hypoperfusion in patients with cerebral hyperperfusion following carotid endarterectomy: SPECT study.

Authors:  Kuniaki Ogasawara; Masakazu Kobayashi; Yasunori Suga; Kohei Chida; Hideo Saito; Nobukazu Komoribayashi; Yasunari Otawara; Akira Ogawa
Journal:  Eur J Nucl Med Mol Imaging       Date:  2007-09-26       Impact factor: 9.236

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