Literature DB >> 14731406

Neglected opportunities in apoptosis research.

J F Kerr1.   

Abstract

Interest in apoptosis is currently growing rapidly, and previous articles in this series in TCB have described a number of recent advances in the field. Certain aspects of the process, however, have been relatively neglected. In this article, John Kerr draws attention to some of them, with the hope that he may indicate potentially productive areas for future research.

Entities:  

Year:  1995        PMID: 14731406     DOI: 10.1016/s0962-8924(00)88940-0

Source DB:  PubMed          Journal:  Trends Cell Biol        ISSN: 0962-8924            Impact factor:   20.808


  5 in total

1.  The small GTPase Cdc42 initiates an apoptotic signaling pathway in Jurkat T lymphocytes.

Authors:  T H Chuang; K M Hahn; J D Lee; D E Danley; G M Bokoch
Journal:  Mol Biol Cell       Date:  1997-09       Impact factor: 4.138

Review 2.  Poly(ADP-ribosyl)ation as a fail-safe, transcription-independent, suicide mechanism in acutely DNA-damaged cells: a hypothesis.

Authors:  A Nagele
Journal:  Radiat Environ Biophys       Date:  1995-11       Impact factor: 1.925

3.  A new nucleolar body appears in Drosophila saltans salivary gland cells before histolysis, in programmed cell death.

Authors:  C C de Oliveira; H E M de Campos Bicudo
Journal:  J Insect Sci       Date:  2006       Impact factor: 1.857

4.  Determining the contents and cell origins of apoptotic bodies by flow cytometry.

Authors:  Lanzhou Jiang; Stephanie Paone; Sarah Caruso; Georgia K Atkin-Smith; Thanh Kha Phan; Mark D Hulett; Ivan K H Poon
Journal:  Sci Rep       Date:  2017-10-31       Impact factor: 4.379

5.  Arginase II downregulates nitric oxide (NO) production and prevents NO-mediated apoptosis in murine macrophage-derived RAW 264.7 cells.

Authors:  T Gotoh; M Mori
Journal:  J Cell Biol       Date:  1999-02-08       Impact factor: 10.539

  5 in total

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