Literature DB >> 12234054

Nitric oxide-induced inhibition of mouse paw edema: involvement of soluble guanylate cyclase and potassium channels.

D Fernandes1, J E Da Silva-Santos, J Assreuy.   

Abstract

OBJECTIVE AND
DESIGN: To investigate the effect of nitric oxide (NO) donors on inflammatory mouse paw edema (MPE).
MATERIALS AND METHODS: Mice previously treated with sodium nitroprusside (SNP; 1.5, 5 and 10 micromol/kg) or S-nitroso-N-acetyl-DL-penicillamine (SNAP; 7, 14 and 28 micromol/kg) were injected with inflammatory mediators in the paw. Paw edema, myeloperoxidase activity and vascular dye leakage were measured.
RESULTS: Pre-treatment with SNP and SNAP (4 h or 12 h) reduced (approximately 50%) MPE induced by carrageenan, dextran sulfate, bradykinin and histamine but not by serotonin. Pre-treatment with SNP also inhibited carrageenan-induced increases in myeloperoxidase activity and vascular dye leakage. Methylene blue blocked the SNP-induced reduction in MPE when injected 30 min before or 2 h after SNP, but not 4 or 6 h after the NO donor. Tetraethylammonium blocked the SNP-induced reduction in MPE if injected 30 min before or 2, 4 or 6 h after SNP.
CONCLUSIONS: NO donors have a long-lasting anti-inflammatory effect in MPE, which involves guanylate cyclase and tetraethylammonium-sensitive potassium channels.

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Year:  2002        PMID: 12234054     DOI: 10.1007/pl00000318

Source DB:  PubMed          Journal:  Inflamm Res        ISSN: 1023-3830            Impact factor:   4.575


  1 in total

1.  S-Nitrosated Polypropylene Sulfide Nanoparticles for Thiol-Dependent Transnitrosation and Toxicity Against Adult Female Filarial Worms.

Authors:  Alex Schudel; Timothy Kassis; J Brandon Dixon; Susan N Thomas
Journal:  Adv Healthc Mater       Date:  2015-05-04       Impact factor: 9.933

  1 in total

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