Literature DB >> 12219030

Heparanase-1 expression is associated with the metastatic potential of breast cancer.

Justin B Maxhimer1, Roderick M Quiros, Robyn Stewart, Kambiz Dowlatshahi, Paolo Gattuso, Ming Fan, Richard A Prinz, Xiulong Xu.   

Abstract

BACKGROUND: Metastasis of malignant breast cells is in part mediated through degradation of the extra-cellular matrix by proteolysis, enabling malignant cells to migrate through the surrounding stroma. Heparanase-1 (HPR1) is an endoglycosidase that specifically degrades the heparan sulfate (HS) moiety of proteoglycans, a component of the extracellular matrix and basement membrane.
METHODS: Fifty-one primary breast tumors, 13 lymph node metastases, 4 ductal carcinoma in situ, 7 benign, and 5 normal specimens were examined for HPR1 expression using immunohistochemical staining. The functional role of HPR1 expression was determined by examining HS deposition using immunofluorescence staining.
RESULTS: Sixteen of 30 breast carcinomas (53%) with sentinel node metastasis expressed HPR1. In contrast, only 5 of 21 nonmetastatic primary breast carcinomas (23%) were HPR1 positive. Eighteen of 30 breast carcinomas between 1 and 5 cm expressed HPR1, compared with 3 of 21 HPR1-positive specimens in tumors < or =1 cm. Statistical analysis revealed that HPR1 expression was associated with breast tumor metastases (P =.04) and primary tumors between 1 and 5 cm (P =.002). Ninety percent of HPR1-positive tumors lacked HS deposition, suggesting an inverse correlation between HPR1 expression and HS deposition.
CONCLUSIONS: HPR1 expression correlates with the lack of HS deposition and with the metastatic potential of breast cancers. The frequency of HPR1 is significantly higher in breast tumors between 1 and 5 cm than in tumors < or =1 cm.

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Year:  2002        PMID: 12219030     DOI: 10.1067/msy.2002.125719

Source DB:  PubMed          Journal:  Surgery        ISSN: 0039-6060            Impact factor:   3.982


  40 in total

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Review 4.  A visual-quantitative analysis of fibroblastic stromagenesis in breast cancer progression.

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Authors:  G Rao; H G Ding; W Huang; D Le; J B Maxhimer; A Oosterhof; T van Kuppevelt; H Lum; E J Lewis; V Reddy; R A Prinz; X Xu
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Review 8.  Mechanisms of heparanase inhibitors in cancer therapy.

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Review 9.  Versatile role of heparanase in inflammation.

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10.  Heparanase mRNA expression and point mutation in hepatocellular carcinoma.

Authors:  Xiao-Peng Chen; Yin-Bib Liu; Jing Rui; Shu-You Peng; Cheng-Hong Peng; Zi-Yan Zhou; Liang-Hui Shi; Hong-Wei Shen; Bin Xu
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