Literature DB >> 12080182

[alpha]-Secretase ADAM10 as well as [alpha]APPs is reduced in platelets and CSF of Alzheimer disease patients.

Francesca Colciaghi1, Barbara Borroni, Lucia Pastorino, Elena Marcello, Martina Zimmermann, Flaminio Cattabeni, Alessandro Padovani, Monica Di Luca.   

Abstract

BACKGROUND: Members of membrane-bound disintegrin metalloproteinases (ADAMs) were shown to be capable of cleaving amyloid precursor protein (APP) at the alpha-cleavage site in different cell systems. One of the candidate alpha-secretases identified in this family is ADAM10. The present study addresses the following major questions: 1) Are the levels of an alpha-secretase candidate (i.e., ADAM10) reduced in accessible cells of Alzheimer Disease (AD) patients? 2) Are ADAM10 levels in the peripheral cells of AD patients related to a concomitant decrease in alpha APPs?
MATERIALS AND METHODS: Western Blot analysis of ADAM10 is performed on platelet homogenates from 33 sporadic AD patients and on 26 age-matched control subjects. Moreover, the levels of alpha-secretase metabolite (alpha APPs) are tested both in platelets and cerebrospinal fluid (CSF) of the same pool of subjects by means of Western blot with a specific antibody.
RESULTS: A significant decrease of platelet ADAM10 levels is observed in patients affected by probable AD when compared to control subjects and this is paralleled by a reduced level of alpha APPs released from platelets. Moreover, in the same pool of AD patients, alpha APPs levels were reduced concomitantly in CSF.
CONCLUSIONS: ADAM10 is expressed in platelets. A reduced level of ADAM10 is observed in platelets obtained from AD patients compared to age-matched controls. Further, in the same pool of AD patients, a qualitatively and quantitatively similar decrease in alpha APPs is present both in thrombin-activated platelets and CSF, thus suggesting that alterations of APP processing might occur both in the neuronal compartment and peripheral cells.

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Year:  2002        PMID: 12080182      PMCID: PMC2039975     

Source DB:  PubMed          Journal:  Mol Med        ISSN: 1076-1551            Impact factor:   6.354


  79 in total

Review 1.  Functions of the APP gene family in the nervous system: insights from mouse models.

Authors:  Dorothee Aydin; Sascha W Weyer; Ulrike C Müller
Journal:  Exp Brain Res       Date:  2011-09-20       Impact factor: 1.972

2.  Translational repression of the disintegrin and metalloprotease ADAM10 by a stable G-quadruplex secondary structure in its 5'-untranslated region.

Authors:  Sven Lammich; Frits Kamp; Judith Wagner; Brigitte Nuscher; Sonja Zilow; Ann-Katrin Ludwig; Michael Willem; Christian Haass
Journal:  J Biol Chem       Date:  2011-11-07       Impact factor: 5.157

Review 3.  Regulation of α-secretase ADAM10 expression and activity.

Authors:  Kristina Endres; Falk Fahrenholz
Journal:  Exp Brain Res       Date:  2011-10-04       Impact factor: 1.972

4.  Amyloid precursor protein (APP) processing genes and cerebrospinal fluid APP cleavage product levels in Alzheimer's disease.

Authors:  L M Bekris; N M Galloway; S Millard; D Lockhart; G Li; D R Galasko; M R Farlow; C M Clark; J F Quinn; J A Kaye; G D Schellenberg; J B Leverenz; P Seubert; D W Tsuang; E R Peskind; C E Yu
Journal:  Neurobiol Aging       Date:  2010-12-31       Impact factor: 4.673

5.  Coated-platelets correlate with disease progression in Alzheimer disease.

Authors:  Calin I Prodan; E D Ross; A S Vincent; G L Dale
Journal:  J Neurol       Date:  2007-03-22       Impact factor: 4.849

Review 6.  Platelets and Alzheimer's disease: Potential of APP as a biomarker.

Authors:  Geneviève Evin; Qiao-Xin Li
Journal:  World J Psychiatry       Date:  2012-12-22

Review 7.  The complex world of post-transcriptional mechanisms: is their deregulation a common link for diseases? Focus on ELAV-like RNA-binding proteins.

Authors:  Alessia Pascale; Stefano Govoni
Journal:  Cell Mol Life Sci       Date:  2011-09-10       Impact factor: 9.261

8.  Association of ADAM10 and CAMK2A polymorphisms with conduct disorder: evidence from family-based studies.

Authors:  Xue-Qiu Jian; Ke-Sheng Wang; Tie-Jian Wu; Joel J Hillhouse; Jerald E Mullersman
Journal:  J Abnorm Child Psychol       Date:  2011-08

9.  Expression of the anti-amyloidogenic secretase ADAM10 is suppressed by its 5'-untranslated region.

Authors:  Sven Lammich; Dominik Buell; Sonja Zilow; Ann-Katrin Ludwig; Brigitte Nuscher; Stefan F Lichtenthaler; Claudia Prinzen; Falk Fahrenholz; Christian Haass
Journal:  J Biol Chem       Date:  2010-03-26       Impact factor: 5.157

10.  Potential late-onset Alzheimer's disease-associated mutations in the ADAM10 gene attenuate {alpha}-secretase activity.

Authors:  Minji Kim; Jaehong Suh; Donna Romano; Mimy H Truong; Kristina Mullin; Basavaraj Hooli; David Norton; Giuseppina Tesco; Kathy Elliott; Steven L Wagner; Robert D Moir; K David Becker; Rudolph E Tanzi
Journal:  Hum Mol Genet       Date:  2009-07-15       Impact factor: 6.150

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