Literature DB >> 12076840

Clobazam shows a different antiepileptic action profile from clonazepam and zonisamide in Ihara epileptic rats.

Yoshiki Miura1, Shigeru Amano, Ryuzo Torii, Nobuo Ihara.   

Abstract

PURPOSE: Clobazam (CLB, 1,5-benzodiazepine, 1,5-BZP) has been reported to show unique antiepileptic action profile distinguishing from standard 1,4-BZPs. To further elucidate the action profile of CLB, its effects on the abnormal circling fits (ACFs) and generalized tonic-clonic convulsions (GTCs) in Ihara epileptic rats (IERs), a genetically epileptic mutant, were examined in comparison with conventional antiepileptic drugs (AEDs), a 1,4-BZP, clonazepam (CZP) and a non-BZP, zonisamide (ZNS).
METHODS: The incidence of ACFs or GTCs in IERs was recorded automatically by the computer-assisted behavior monitoring system (COBAS N-IV) before, during and after the drug treatment period for 5 days in each. The drugs were orally administered twice daily. The daily and total incidences of ACFs or GTCs were calculated every each period in each dose group. The incidences of various behaviors such as feeding, gnawing and scratching recorded simultaneously were used for evaluating the behavioral activity (BA).
RESULTS: CLB (30 and 60 mg/kg) prevented the appearance of ACFs and GTCs without affecting BAs. CZP (1 and 3 mg/kg) suppressed the occurrence of ACFs but induced no effect on the incidence of GTCs. Furthermore, it inhibited BAs at the same doses. ZNS (15 mg/kg) suppressed GTCs but little ACFs without affecting BA.
CONCLUSION: CLB exhibited a different action profile from CZP and ZNS in a novel epileptic mutant, IERs, and was expected to be a useful AED superior to 1,4-BZPs in clinical practice.

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Year:  2002        PMID: 12076840     DOI: 10.1016/s0920-1211(02)00032-3

Source DB:  PubMed          Journal:  Epilepsy Res        ISSN: 0920-1211            Impact factor:   3.045


  5 in total

1.  Clobazam as an adjunctive therapy in treating seizures associated with Lennox-Gastaut syndrome.

Authors:  Jennifer T Leahy; Catherine J Chu-Shore; Janet L Fisher
Journal:  Neuropsychiatr Dis Treat       Date:  2011-11-11       Impact factor: 2.570

Review 2.  Advances on genetic rat models of epilepsy.

Authors:  Tadao Serikawa; Tomoji Mashimo; Takashi Kuramoro; Birger Voigt; Yukihiro Ohno; Masashi Sasa
Journal:  Exp Anim       Date:  2014-10-14

3.  Functional characterization of the 1,5-benzodiazepine clobazam and its major active metabolite N-desmethylclobazam at human GABA(A) receptors expressed in Xenopus laevis oocytes.

Authors:  Harriet Hammer; Bjarke Ebert; Henrik Sindal Jensen; Anders A Jensen
Journal:  PLoS One       Date:  2015-03-23       Impact factor: 3.240

4.  The clobazam metabolite N-desmethyl clobazam is an α2 preferring benzodiazepine with an improved therapeutic window for antihyperalgesia.

Authors:  William T Ralvenius; Mario A Acuña; Dietmar Benke; Alain Matthey; Youssef Daali; Uwe Rudolph; Jules Desmeules; Hanns Ulrich Zeilhofer; Marie Besson
Journal:  Neuropharmacology       Date:  2016-07-05       Impact factor: 5.250

5.  Clobazam and its active metabolite N-desmethylclobazam display significantly greater affinities for α₂- versus α₁-GABA(A)-receptor complexes.

Authors:  Henrik Sindal Jensen; Kathryn Nichol; Deborah Lee; Bjarke Ebert
Journal:  PLoS One       Date:  2014-02-12       Impact factor: 3.240

  5 in total

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