Literature DB >> 11867603

Long-term effects of light damage on the retina of albino and pigmented rats.

Marguerite Wasowicz1, Cécile Morice, Patricia Ferrari, Jacques Callebert, Claudine Versaux-Botteri.   

Abstract

PURPOSE: To observe the morphology and physiology of the retina in rats 11 weeks after a constant (24-hour) but moderate (500-lux) illumination for 1 week.
METHODS: Levels of aspartate, gamma-aminobutyric acid (GABA), glutamate, glutamine, and taurine were measured by high-pressure liquid chromatography (HPLC) in the retina and vitreous humor of albino (Wistar) and pigmented (Long-Evans) rats. Semithin sections were used to determine retinal morphology. The TUNEL method was used to detect cells degenerating by apoptosis. Because the GABAergic system has been shown to be particularly sensitive to the loss of photoreceptors, an additional immunohistochemical study using anti-GABA, anti-glutamate decarboxylase (GAD)(67) and anti-GAD(65) antibodies was performed.
RESULTS: No apparent morphologic changes were found in the retina of pigmented rats after constant illumination, whereas in albino rats disappearance of photoreceptors (except in the extreme retinal periphery) and cell bodies was observed. A significant number of TUNEL-positive nuclei also occurred in the remaining nuclear and ganglion cell layers. However, no change in the distribution of GABA, GAD(67), and GAD(65) immunoreactivities was found in either strain under constant illumination compared with control animals. Constant illumination affected the retinal levels of aspartate, glutamate, glutamine, glycine in both strains, whereas GABA contents did not change and taurine was decreased only in albino rats. A significant increase of vitreal glutamate levels was also found in both strains and of taurine levels only in albino rats.
CONCLUSIONS: Phototoxicity can provoke durable retinal alterations beyond the period of lighting, suggesting progressive and probably continuous modifications of retinal physiology, even in pigmented animals in which the retina seems morphologically normal.

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Year:  2002        PMID: 11867603

Source DB:  PubMed          Journal:  Invest Ophthalmol Vis Sci        ISSN: 0146-0404            Impact factor:   4.799


  22 in total

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Journal:  FASEB J       Date:  2011-09-22       Impact factor: 5.191

2.  Expression patterns of iron regulatory proteins after intense light exposure in a cone-dominated retina.

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Journal:  FASEB J       Date:  2018-01-24       Impact factor: 5.191

4.  Phototoxic effects of commercial photographic flash lamp on rat eyes.

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7.  Cell Death Pathways in Mutant Rhodopsin Rat Models Identifies Genotype-Specific Targets Controlling Retinal Degeneration.

Authors:  Ishaq A Viringipurampeer; Cheryl Y Gregory-Evans; Andrew L Metcalfe; Emran Bashar; Orson L Moritz; Kevin Gregory-Evans
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8.  Changes in the synaptoarchitectonics of the retina after light-induced damage and their correction with antioxidants of plant origin.

Authors:  S V Logvinov; E Yu Varakuta; A A Zhdankina; A V Potapov; M B Plotnikov
Journal:  Neurosci Behav Physiol       Date:  2009-01-13

Review 9.  Age-related retinal degeneration in animal models of aging: possible involvement of taurine deficiency and oxidative stress.

Authors:  Julius Militante; John B Lombardini
Journal:  Neurochem Res       Date:  2004-01       Impact factor: 3.996

10.  Retinal ganglion cell axonal compression by retinal vessels in light-induced retinal degeneration.

Authors:  Diego García-Ayuso; Manuel Salinas-Navarro; Marta Agudo-Barriuso; Luis Alarcón-Martínez; Manuel Vidal-Sanz; María P Villegas-Pérez
Journal:  Mol Vis       Date:  2011-06-25       Impact factor: 2.367

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