Literature DB >> 11849244

Clinical experience of amiodarone-induced thyrotoxicosis over a 3-year period: role of colour-flow Doppler sonography.

S E M Eaton1, H A Euinton, C M Newman, A P Weetman, W M Bennet.   

Abstract

OBJECTIVE: Current thinking is that amiodarone-induced thyrotoxicosis (AIT) might be either iodine-induced thyrotoxicosis in latent hyperthyroidism (Type 1) or destructive thyroiditis (Type 2), and also that colour-flow Doppler sonography (CFDS) of the thyroid and serum interleukin 6 (IL-6) are tools that can classify AIT and direct treatment. To assess the validity of this thinking, our objective was to determine whether CFDS and IL-6 identified AIT subgroups with distinct features.
DESIGN: Retrospective case-note audit of all patients presenting with AIT to the Endocrine Department of a UK teaching hospital over a 3-year period. To assess proportions of Type 1 vs. Type 2 AIT and to compare and contrast their clinical features. PATIENTS: 37 patients were identified with AIT (mean age 65, range 20-86 years). In 30 patients in whom AIT persisted, 25 underwent CFDS.
RESULTS: In 25 patients who underwent CFDS, 10 (40%) were classified as Type 1, 10 (40%) as Type 2 and 5 (20%) as indeterminate type. In the patients classified by CFDS in whom AIT persisted, 40% of Type 1 patients were male vs. 90% of Type 2 patients. Also, free T4 tended to be lower in patients presenting with Type 1 AIT (52.1 +/- 7.5 pmol/l) compared to Type 2 (74.8 +/- 8.1 pmol/l, P = 0.08), free T3 was lower (8.8 +/- 0.9 vs. 15.6 +/- 3.0 pmol/l, P = 0.03) and the cumulative amiodarone dose was lower (66 +/- 20 vs. 186 +/- 28 g, P = 0.002). We used less prednisolone to achieve euthyroidism in patients with Type 1 AIT whereas carbimazole doses were not different and the time to euthyroidism was the same in both groups (81 +/- 21 vs. 88 +/- 13 days). IL-6 was raised in two patients with Type 1 and in one patient with Type 2 AIT.
CONCLUSIONS: CFDS could characterize two distinct subtypes in patients with AIT. Conversely, IL-6 seemed to be an unhelpful test in this context.

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Year:  2002        PMID: 11849244     DOI: 10.1046/j.0300-0664.2001.01457.x

Source DB:  PubMed          Journal:  Clin Endocrinol (Oxf)        ISSN: 0300-0664            Impact factor:   3.478


  18 in total

1.  The presence of anti-thyroglobulin (TgAb) and/or anti-thyroperoxidase antibodies (TPOAb) does not exclude the diagnosis of type 2 amiodarone-induced thyrotoxicosis.

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Review 2.  [Thyroid gland and the heart : Pathophysiological background, diagnostic and therapeutic consequences].

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3.  Role of color Doppler in differentiation of Graves' disease and thyroiditis in thyrotoxicosis.

Authors:  Ragab Hani Donkol; Aml Mohamed Nada; Sami Boughattas
Journal:  World J Radiol       Date:  2013-04-28

Review 4.  Amiodarone and the thyroid: a 2012 update.

Authors:  F Bogazzi; L Tomisti; L Bartalena; F Aghini-Lombardi; E Martino
Journal:  J Endocrinol Invest       Date:  2012-03-19       Impact factor: 4.256

5.  Management of amiodarone-related thyroid problems.

Authors:  Shashithej K Narayana; David R Woods; Christopher J Boos
Journal:  Ther Adv Endocrinol Metab       Date:  2011-06       Impact factor: 3.565

6.  Long-term outcome of thyroid function after amiodarone-induced thyrotoxicosis, as compared to subacute thyroiditis.

Authors:  F Bogazzi; E Dell'Unto; M L Tanda; L Tomisti; C Cosci; F Aghini-Lombardi; C Sardella; A Pinchera; L Bartalena; E Martino
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7.  A Case of Amiodarone-Induced Thyrotoxicosis: A diagnostic and therapeutic dilemma.

Authors:  Ps Sudheer Ahamed; Abe Mathew
Journal:  Sultan Qaboos Univ Med J       Date:  2009-12-19

8.  Thyroid color flow doppler sonography and radioiodine uptake in 55 consecutive patients with amiodarone-induced thyrotoxicosis.

Authors:  F Bogazzi; E Martino; E Dell'Unto; S Brogioni; C Cosci; F Aghini-Lombardi; C Ceccarelli; A Pinchera; L Bartalena; L E Braverman
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Review 9.  Effects of amiodarone therapy on thyroid function.

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Review 10.  Amiodarone-induced thyrotoxicosis: a review.

Authors:  Wendy Tsang; Robyn L Houlden
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