Literature DB >> 11823668

Apolipoprotein e genotype and outcome in aneurysmal subarachnoid hemorrhage.

Clarence H S Leung1, W S Poon, L M Yu, George K C Wong, H K Ng.   

Abstract

BACKGROUND AND
PURPOSE: Active management of ruptured intracranial aneurysm in subarachnoid hemorrhage is indicated in patients with favorable prognosis. Outcome prediction is based on patient characteristics and clinical and radiological factors. Current clinical grading scales are imprecise, with low interobserver reproducibility. Therefore, outcome prediction remains inconsistent and decision making becomes difficult, especially for patients with poor clinical grade.
METHODS: The possible relationship between apolipoprotein E genotype and the outcome of patients suffering spontaneous subarachnoid hemorrhage was investigated. A prospective study was conducted on all patients with spontaneous aneurysmal subarachnoid hemorrhage admitted to our unit during a 2-year period. All patients were managed according to standard protocol, and treatments were given according to their clinical grading. Patient characteristics, clinical grade, radiological grade, and apolipoprotein E genotype were documented. The focus of the study was the 6-month neurological outcome for this group of patients after they were discharged.
RESULTS: Seventy-two patients with aneurysmal subarachnoid hemorrhage were admitted to the Prince of Wales Hospital in Shatin, Hong Kong, China, from February 1998 to February 2000. Their ages ranged from 24 to 95 years of age, with a mean (SD) age of 58.3 (15.0) years. Apolipoprotein E epsilon4 was found in 15 patients (21%). At 6 months, Glasgow Outcome Scale score < or = 3 was found in 29 patients (40%). Univariate analysis showed that older patients (odds ratio [OR], 1.03; 95% CI, 1.00 to 1.07; P=0.07) and patients with poor Fisher's grade (OR, 4.5; 95% CI, 1.3 to 15.2; P=0.01), poor World Federation of Neurological Surgeons grade (OR, 5.8; 95% CI, 1.9 to 17.8; P=0.002), or apolipoprotein E epsilon4 (OR, 6.0; 95% CI, 1.7 to 21.3; P=0.006) were more likely to attain unfavorable outcome at 6 months. The additional effect of apolipoprotein E epsilon4 remained significant in the multiple logistic regression model (OR, 11.3; 95% CI, 2.2 to 57.0; P=0.003); the gain in predictive performance was not significant (P=0.26).
CONCLUSIONS: Apolipoprotein E epsilon4 genotype is related to poor outcome in patients with subarachnoid hemorrhage.

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Year:  2002        PMID: 11823668     DOI: 10.1161/hs0202.102326

Source DB:  PubMed          Journal:  Stroke        ISSN: 0039-2499            Impact factor:   7.914


  15 in total

1.  Apolipoprotein E epsilon 2 is associated with new hemorrhage risk in brain arteriovenous malformations.

Authors:  Ludmila Pawlikowska; K Y Trudy Poon; Achal S Achrol; Charles E McCulloch; Connie Ha; Kristen Lum; Jonathan G Zaroff; Nerissa U Ko; S Claiborne Johnston; Stephen Sidney; Douglas A Marchuk; Michael T Lawton; Pui-Yan Kwok; William L Young
Journal:  Neurosurgery       Date:  2006-05       Impact factor: 4.654

2.  Genes and outcome after aneurysmal subarachnoid haemorrhage.

Authors:  Y M Ruigrok; A J C Slooter; A Bardoel; C J M Frijns; G J E Rinkel; C Wijmenga
Journal:  J Neurol       Date:  2005-02-23       Impact factor: 4.849

Review 3.  Effects of apolipoprotein E genotype on outcome after ischaemic stroke, intracerebral haemorrhage and subarachnoid haemorrhage.

Authors:  N A Martínez-González; C L M Sudlow
Journal:  J Neurol Neurosurg Psychiatry       Date:  2006-08-22       Impact factor: 10.154

4.  Association of tumor necrosis factor-alpha-238G>A and apolipoprotein E2 polymorphisms with intracranial hemorrhage after brain arteriovenous malformation treatment.

Authors:  Achal S Achrol; Helen Kim; Ludmila Pawlikowska; K Y Trudy Poon; Charles E McCulloch; Nerissa U Ko; S Claiborne Johnston; Michael W McDermott; Jonathan G Zaroff; Michael T Lawton; Pui-Yan Kwok; William L Young
Journal:  Neurosurgery       Date:  2007-10       Impact factor: 4.654

5.  Multiplexed protein profiling after aneurysmal subarachnoid hemorrhage: characterization of differential expression patterns in cerebral vasospasm.

Authors:  Brian P Walcott; Anoop P Patel; Christopher J Stapleton; Rikin A Trivedi; Adam M H Young; Christopher S Ogilvy
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6.  Association of APOE e4 and cerebrovascular pathology in traumatic brain injury.

Authors:  C Smith; D I Graham; L S Murray; J Stewart; J A R Nicoll
Journal:  J Neurol Neurosurg Psychiatry       Date:  2006-03       Impact factor: 10.154

7.  A novel apoE-derived therapeutic reduces vasospasm and improves outcome in a murine model of subarachnoid hemorrhage.

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Journal:  Neurocrit Care       Date:  2006       Impact factor: 3.210

Review 8.  Genetic determinants of cerebral vasospasm, delayed cerebral ischemia, and outcome after aneurysmal subarachnoid hemorrhage.

Authors:  Andrew F Ducruet; Paul R Gigante; Zachary L Hickman; Brad E Zacharia; Eric J Arias; Bartosz T Grobelny; Justin W Gorski; Stephan A Mayer; E Sander Connolly
Journal:  J Cereb Blood Flow Metab       Date:  2010-01-13       Impact factor: 6.200

Review 9.  Biomarkers as outcome predictors in subarachnoid hemorrhage--a systematic review.

Authors:  Caron M Hong; Cigdem Tosun; David B Kurland; Volodymyr Gerzanich; David Schreibman; J Marc Simard
Journal:  Biomarkers       Date:  2014-02-05       Impact factor: 2.658

10.  APOE genotype and functional outcome following aneurysmal subarachnoid hemorrhage.

Authors:  Matthew J Gallek; Yvette P Conley; Paula R Sherwood; Michael B Horowitz; Amin Kassam; Sheila A Alexander
Journal:  Biol Res Nurs       Date:  2008-11-17       Impact factor: 2.522

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