Literature DB >> 11530003

Does acute hypoxia cause fetal arterial blood flow redistribution?

E Krampl1, K Chalubinski, C Schatten, P Husslein.   

Abstract

Chronic fetal hypoxia in fetal growth restriction due to impaired placentation is associated with centralization of blood flow to the vital organs, such as brain, heart and adrenal glands, in order to maintain oxygenation. There is a correlation between fetal hypoxemia and low impedance to blood flow in the middle cerebral artery. However, there is no association between abnormal flow velocity waveforms and fetal distress in an unselected population, and this reported case also suggests that Doppler ultrasound is of no value in identifying acute fetal distress.

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Year:  2001        PMID: 11530003     DOI: 10.1046/j.1469-0705.2001.00501.x

Source DB:  PubMed          Journal:  Ultrasound Obstet Gynecol        ISSN: 0960-7692            Impact factor:   7.299


  3 in total

1.  Formation of the neocortex in rats after prenatal hypoxia.

Authors:  L I Khozhai; V A Otellin; V B Kostkin
Journal:  Neurosci Behav Physiol       Date:  2004-02

2.  Associations between arterial oxygen saturation, body size and limb measurements among high-altitude Andean children.

Authors:  Emma Pomeroy; Jay T Stock; Sanja Stanojevic; J Jaime Miranda; Tim J Cole; Jonathan C K Wells
Journal:  Am J Hum Biol       Date:  2013-08-01       Impact factor: 1.937

3.  Behaviour of the Foramen Ovale Flow in Fetuses with Intrauterine Growth Restriction.

Authors:  Ângela R L Nader; Paulo Zielinsky; Alexandre Antonio Naujorks; Luiz Henrique S Nicoloso; Antonio Luiz Piccoli Junior; Natássia Miranda Sulis; Luiza Ferreira van der Sand; Victoria de Bittencourt Antunes; Gabriela Dos Santos Marinho; Fernanda Greinert Dos Santos; Natan Pereira Gosmann; Eduardo Becker Júnior; Renato Frajndlich; Tamara Beherens; Marcelo Brandão da Silva; Carolina Barbisan; Stefano Busato; Mauro Lopes; Caroline Klein
Journal:  Obstet Gynecol Int       Date:  2018-01-17
  3 in total

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