Literature DB >> 11389469

Living dangerously: how Helicobacter pylori survives in the human stomach.

C Montecucco1, R Rappuoli.   

Abstract

Helicobacter pylori was already present in the stomach of primitive humans as they left Africa and spread through the world. Today, it still chronically infects more than 50% of the human population, causing, in some cases, severe diseases such as peptic ulcers and stomach cancer. To succeed in these long-term associations, H. pylori has developed a unique set of virulence factors, which allow survival in a unique and hostile ecological niche--the human stomach.

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Year:  2001        PMID: 11389469     DOI: 10.1038/35073084

Source DB:  PubMed          Journal:  Nat Rev Mol Cell Biol        ISSN: 1471-0072            Impact factor:   94.444


  145 in total

1.  How the loop and middle regions influence the properties of Helicobacter pylori VacA channels.

Authors:  F Tombola; C Pagliaccia; S Campello; J L Telford; C Montecucco; E Papini; M Zoratti
Journal:  Biophys J       Date:  2001-12       Impact factor: 4.033

2.  Associations of TNF-A-1031TT and -857TT genotypes with Helicobacter pylori seropositivity and gastric atrophy among Japanese Brazilians.

Authors:  Yoshiko Atsuta; Lucy S Ito; Sueli M Oba-Shinjo; Miyuki Uno; Samuel Katsuyuki Shinjo; Suely K N Marie; Yasuyuki Goto; Nobuyuki Hamajima
Journal:  Int J Clin Oncol       Date:  2006-04       Impact factor: 3.402

3.  cag+ Helicobacter pylori induces homotypic aggregation of macrophage-like cells by up-regulation and recruitment of intracellular adhesion molecule 1 to the cell surface.

Authors:  Stefan Moese; Matthias Selbach; Thomas F Meyer; Steffen Backert
Journal:  Infect Immun       Date:  2002-08       Impact factor: 3.441

4.  Expression of Helicobacter pylori vacuolating toxin in Escherichia coli.

Authors:  Mark S McClain; Timothy L Cover
Journal:  Infect Immun       Date:  2003-04       Impact factor: 3.441

5.  Global transposon mutagenesis and essential gene analysis of Helicobacter pylori.

Authors:  Nina R Salama; Benjamin Shepherd; Stanley Falkow
Journal:  J Bacteriol       Date:  2004-12       Impact factor: 3.490

6.  Peptidoglycan crosslinking relaxation promotes Helicobacter pylori's helical shape and stomach colonization.

Authors:  Laura K Sycuro; Zachary Pincus; Kimberley D Gutierrez; Jacob Biboy; Chelsea A Stern; Waldemar Vollmer; Nina R Salama
Journal:  Cell       Date:  2010-05-28       Impact factor: 41.582

7.  Activation of the Raf-MEK-ERK pathway is required for neutrophil extracellular trap formation.

Authors:  Abdul Hakkim; Tobias A Fuchs; Nancy E Martinez; Simone Hess; Heino Prinz; Arturo Zychlinsky; Herbert Waldmann
Journal:  Nat Chem Biol       Date:  2010-12-19       Impact factor: 15.040

8.  Mouse Models Of Helicobacter Infection And Gastric Pathologies.

Authors:  Kimberley D'Costa; Michelle Chonwerawong; Le Son Tran; Richard L Ferrero
Journal:  J Vis Exp       Date:  2018-10-18       Impact factor: 1.355

9.  Interaction with CagF is required for translocation of CagA into the host via the Helicobacter pylori type IV secretion system.

Authors:  Marc Roger Couturier; Elizabetta Tasca; Cesare Montecucco; Markus Stein
Journal:  Infect Immun       Date:  2006-01       Impact factor: 3.441

10.  Epidemiological link between gastric disease and polymorphisms in VacA and CagA.

Authors:  Sungil Jang; Kathleen R Jones; Cara H Olsen; Young Min Joo; Yun-Jung Yoo; In-Sik Chung; Jeong-Heon Cha; D Scott Merrell
Journal:  J Clin Microbiol       Date:  2009-12-02       Impact factor: 5.948

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