Literature DB >> 11176967

Analysis of cerebral shape in Williams syndrome.

J E Schmitt1, S Eliez, U Bellugi, A L Reiss.   

Abstract

BACKGROUND: As a neurobehavioral disorder with a specific neurocognitive profile and a well-defined genetic etiology, Williams syndrome (WMS) provides an exceptional opportunity to examine associations among measures of behavior, neuroanatomy, and genetics. This study was designed to determine how cerebral shape differs between the brains of subjects with WMS and those of normal controls.
SUBJECTS: Twenty adults with clinically and genetically diagnosed WMS (mean +/- SD age, 28.5 +/- 8.3 years) and 20 healthy, age- and sex-matched controls (mean +/- SD age, 28.5 +/- 8.2 years).
DESIGN: High-resolution structural magnetic resonance imaging data were used for shape-based morphological analysis of the right and left cerebral hemispheres and the corpus callosum. Statistical analyses were performed to examine group differences.
RESULTS: Both right and left cerebral hemispheres of subjects with WMS bend to a lesser degree in the sagittal plane than normal controls (P<.001). The corpus callosum also bends less in subjects with WMS (P =.05). In addition, subjects with WMS have decreased cerebral (P<.001) and corpus callosum (P<.001) midline lengths.
CONCLUSIONS: Subjects with WMS have significantly different cerebral shape from normal controls, perhaps due to decreased parieto-occipital lobe volumes relative to frontal regions. The similar observation in the corpus callosum may be associated with a decreased size of the splenium in WMS. These findings may provide important clues to the effect of genes in the WMS-deleted region on brain development.

Entities:  

Mesh:

Year:  2001        PMID: 11176967     DOI: 10.1001/archneur.58.2.283

Source DB:  PubMed          Journal:  Arch Neurol        ISSN: 0003-9942


  20 in total

1.  Corpus callosum morphology and ventricular size in chromosome 22q11.2 deletion syndrome.

Authors:  Alexei M C Machado; Tony J Simon; Vy Nguyen; Donna M McDonald-McGinn; Elaine H Zackai; James C Gee
Journal:  Brain Res       Date:  2006-12-13       Impact factor: 3.252

2.  Callosal morphology in Williams syndrome: a new evaluation of shape and thickness.

Authors:  Eileen Luders; Margherita Di Paola; Francesco Tomaiuolo; Paul M Thompson; Arthur W Toga; Stefano Vicari; Michael Petrides; Carlo Caltagirone
Journal:  Neuroreport       Date:  2007-02-12       Impact factor: 1.837

3.  Retinotopically defined primary visual cortex in Williams syndrome.

Authors:  Rosanna K Olsen; J Shane Kippenhan; Shruti Japee; Philip Kohn; Carolyn B Mervis; Ziad S Saad; Colleen A Morris; Andreas Meyer-Lindenberg; Karen Faith Berman
Journal:  Brain       Date:  2009-03-02       Impact factor: 13.501

Review 4.  Insights into brain development from neurogenetic syndromes: evidence from fragile X syndrome, Williams syndrome, Turner syndrome and velocardiofacial syndrome.

Authors:  E Walter; P K Mazaika; A L Reiss
Journal:  Neuroscience       Date:  2009-04-17       Impact factor: 3.590

5.  MRI assessment of superior temporal gyrus in Williams syndrome.

Authors:  Adriana Sampaio; Nuno Sousa; Montse Férnandez; Cristiana Vasconcelos; Martha E Shenton; Oscar F Gonçalves
Journal:  Cogn Behav Neurol       Date:  2008-09       Impact factor: 1.600

Review 6.  Bridging the gene-behavior divide through neuroimaging deletion syndromes: Velocardiofacial (22q11.2 Deletion) and Williams (7q11.23 Deletion) syndromes.

Authors:  Daniel Paul Eisenberg; Mbemba Jabbi; Karen Faith Berman
Journal:  Neuroimage       Date:  2010-03-03       Impact factor: 6.556

7.  PTPN11 Gain-of-Function Mutations Affect the Developing Human Brain, Memory, and Attention.

Authors:  Emily M Johnson; Alexandra D Ishak; Paige E Naylor; David A Stevenson; Allan L Reiss; Tamar Green
Journal:  Cereb Cortex       Date:  2019-07-05       Impact factor: 5.357

8.  Leftward lateralization of auditory cortex underlies holistic sound perception in Williams syndrome.

Authors:  Martina Wengenroth; Maria Blatow; Martin Bendszus; Peter Schneider
Journal:  PLoS One       Date:  2010-08-23       Impact factor: 3.240

Review 9.  Wnt signaling in development and disease.

Authors:  Jennifer L Freese; Darya Pino; Samuel J Pleasure
Journal:  Neurobiol Dis       Date:  2009-09-16       Impact factor: 5.996

10.  Intracisternal Gtf2i Gene Therapy Ameliorates Deficits in Cognition and Synaptic Plasticity of a Mouse Model of Williams-Beuren Syndrome.

Authors:  Cristina Borralleras; Ignasi Sahun; Luis A Pérez-Jurado; Victoria Campuzano
Journal:  Mol Ther       Date:  2015-07-28       Impact factor: 11.454

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