Literature DB >> 11101735

Polyvinylchloride infusion lines expose infants to large amounts of toxic plasticizers.

S Loff1, F Kabs, K Witt, J Sartoris, B Mandl, K H Niessen, K L Waag.   

Abstract

PURPOSE: The purpose of this study was to evaluate whether infusion lines are able to leach plasticizers in substantial amounts and thus be a candidate substance for hepatotoxic effects during long-term total parenteral nutrition (TPN).
METHODS: TPN solutions, blood products, and selected drugs typical for preterm infants concerning amount, content, and infusion time were perfused through common polyvinylchloride (PVC) infusion lines. Concentration of diethylhexyl-phthalate (DEHP) before and after perfusion was determined by gas chromatography/mass spectrometry.
RESULTS: Daily quantities of DEHP by 24-hour infusions were Lipid emulsion 20%: 10185.6 microg; aminoacid/glucose-solution: 116.2 microg; midazolaminfusion for sedation: 26.4 microg; fentanyl for sedation: 132.5 microg; propofol for sedation: 6561.0 microg. The amount of DEHP by single doses of blood products (20 mL) were packed red blood cells: 144-608 microg; platelet rich plasma: 928 microg; and fresh frozen plasma: 552-8108 microg. The dose of DEHP for a typical preterm neonate requiring TPN and additional therapy like sedation or blood products is at minimum 10 mg and can easily reach 20 mg/d.
CONCLUSION: This large amount of DEHP is especially disturbing, because it effects the most vulnerable patients (neonates). Whether there is a relation to TPN-induced hepatobiliary dysfunction remains to be elucidated and is under investigation. With respect to recent literature, a biological effect of these doses must be assumed.

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Year:  2000        PMID: 11101735     DOI: 10.1053/jpsu.2000.19249

Source DB:  PubMed          Journal:  J Pediatr Surg        ISSN: 0022-3468            Impact factor:   2.545


  25 in total

1.  Gene expression profiling of DEHP-treated cardiomyocytes reveals potential causes of phthalate arrhythmogenicity.

Authors:  Nikki Gillum Posnack; Norman H Lee; Ronald Brown; Narine Sarvazyan
Journal:  Toxicology       Date:  2010-10-08       Impact factor: 4.221

2.  Phthalate esters affect maturation and function of primate testis tissue ectopically grafted in mice.

Authors:  Jose R Rodriguez-Sosa; Alla Bondareva; Lin Tang; Gleide F Avelar; Krysta M Coyle; Mark Modelski; Whitney Alpaugh; Alan Conley; Katherine Wynne-Edwards; Luiz R França; Stuart Meyers; Ina Dobrinski
Journal:  Mol Cell Endocrinol       Date:  2014-10-27       Impact factor: 4.102

3.  Regulation of arcuate genes by developmental exposures to endocrine-disrupting compounds in female rats.

Authors:  Troy A Roepke; Jennifer A Yang; Ali Yasrebi; Kyle J Mamounis; Elif Oruc; Aparna Mahakali Zama; Mehmet Uzumcu
Journal:  Reprod Toxicol       Date:  2016-04-19       Impact factor: 3.143

Review 4.  Of mice and men (and rats): phthalate-induced fetal testis endocrine disruption is species-dependent.

Authors:  Kamin J Johnson; Nicholas E Heger; Kim Boekelheide
Journal:  Toxicol Sci       Date:  2012-06-14       Impact factor: 4.849

5.  Molecular mechanisms mediating the effect of mono-(2-ethylhexyl) phthalate on hormone-stimulated steroidogenesis in MA-10 mouse tumor Leydig cells.

Authors:  Jinjiang Fan; Kassim Traore; Wenping Li; Hakima Amri; Hongzhan Huang; Cathy Wu; Haolin Chen; Barry Zirkin; Vassilios Papadopoulos
Journal:  Endocrinology       Date:  2010-05-12       Impact factor: 4.736

6.  In utero exposure to the antiandrogen di-(2-ethylhexyl) phthalate decreases adrenal aldosterone production in the adult rat.

Authors:  Daniel B Martinez-Arguelles; Theodore Guichard; Martine Culty; Barry R Zirkin; Vassilios Papadopoulos
Journal:  Biol Reprod       Date:  2011-03-09       Impact factor: 4.285

Review 7.  The adverse cardiac effects of Di(2-ethylhexyl)phthalate and Bisphenol A.

Authors:  Nikki Gillum Posnack
Journal:  Cardiovasc Toxicol       Date:  2014-12       Impact factor: 3.231

8.  Oxidative stress and phthalate-induced down-regulation of steroidogenesis in MA-10 Leydig cells.

Authors:  Liang Zhou; Matthew C Beattie; Chieh-Yin Lin; June Liu; Kassim Traore; Vassilios Papadopoulos; Barry R Zirkin; Haolin Chen
Journal:  Reprod Toxicol       Date:  2013-08-19       Impact factor: 3.143

9.  Clinically relevant concentrations of di (2-ethylhexyl) phthalate (DEHP) uncouple cardiac syncytium.

Authors:  Nikki Gillum; Zaruhi Karabekian; Luther M Swift; Ronald P Brown; Matthew W Kay; Narine Sarvazyan
Journal:  Toxicol Appl Pharmacol       Date:  2009-01-22       Impact factor: 4.219

10.  In utero exposure to di-(2-ethylhexyl) phthalate decreases mineralocorticoid receptor expression in the adult testis.

Authors:  D B Martinez-Arguelles; M Culty; B R Zirkin; V Papadopoulos
Journal:  Endocrinology       Date:  2009-10-09       Impact factor: 4.736

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