Literature DB >> 10828757

Is there a common mechanism for the progression of different types of renal diseases other than proteinuria? Towards the unifying theme of chronic hypoxia.

L G Fine1, D Bandyopadhay, J T Norman.   

Abstract

The question of why chronic renal diseases progress is a topic only recently investigated. Putative causes such as proteinuria do not account for all aspects of progressive renal disease. An alternative mechanism, chronic hypoxia, is proposed that might better explain certain elements of progressive renal disease, but elements of the hypothesis remain subject to further study.

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Year:  2000        PMID： 10828757

Source DB:  PubMed          Journal:  Kidney Int Suppl        ISSN： 0098-6577            Impact factor:   10.545


  88 in total

1.  Predictors of successful completion of diagnostic home sleep testing in patients with chronic kidney disease.

Authors:  Jonathan Lee; Tanvir C Turin; David D M Nicholl; Sofia B Ahmed; Andrea H S Loewen; Brenda R Hemmelgarn; Abul K Azad; Patrick J Hanly
Journal:  Sleep Breath       Date:  2014-11-05       Impact factor: 2.816

Review 2.  The suffocating kidney: tubulointerstitial hypoxia in end-stage renal disease.

Authors:  Imari Mimura; Masaomi Nangaku
Journal:  Nat Rev Nephrol       Date:  2010-09-28       Impact factor: 28.314

3.  Peritubular ischemia contributes more to tubular damage than proteinuria in immune-mediated glomerulonephritis.

Authors:  Muh Geot Wong; Yusuke Suzuki; Chiaki Tanifuji; Hisaya Akiba; Ko Okumura; Takeshi Sugaya; Tokunori Yamamoto; Satoshi Horikoshi; Si Yen Tan; Carol Pollock; Yasuhiko Tomino
Journal:  J Am Soc Nephrol       Date:  2007-12-19       Impact factor: 10.121

4.  Low oxygen stimulates the immune system.

Authors:  J S Maltzman; V H Haase
Journal:  Kidney Int       Date:  2008-04       Impact factor: 10.612

Review 5.  Modeling transport in the kidney: investigating function and dysfunction.

Authors:  Aurélie Edwards
Journal:  Am J Physiol Renal Physiol       Date:  2009-11-04

Review 6.  Anaemia in diabetes: Is there a rationale to TREAT?

Authors:  M C Thomas; M E Cooper; K Rossing; H H Parving
Journal:  Diabetologia       Date:  2006-04-04       Impact factor: 10.122

7.  Hypoxia in renal disease with proteinuria and/or glomerular hypertension.

Authors:  Tetsuhiro Tanaka; Toshio Miyata; Reiko Inagi; Toshiro Fujita; Masaomi Nangaku
Journal:  Am J Pathol       Date:  2004-12       Impact factor: 4.307

8.  Stable expression of HIF-1alpha in tubular epithelial cells promotes interstitial fibrosis.

Authors:  Kuniko Kimura; Masayuki Iwano; Debra F Higgins; Yukinari Yamaguchi; Kimihiko Nakatani; Koji Harada; Atsushi Kubo; Yasuhiro Akai; Erinn B Rankin; Eric G Neilson; Volker H Haase; Yoshihiko Saito
Journal:  Am J Physiol Renal Physiol       Date:  2008-07-30

9.  Loss of Renal Peritubular Capillaries in Hypertensive Patients Is Detectable by Urinary Endothelial Microparticle Levels.

Authors:  In O Sun; Adrian Santelli; Abdelrhman Abumoawad; Alfonso Eirin; Christopher M Ferguson; John R Woollard; Amir Lerman; Stephen C Textor; Amrutesh S Puranik; Lilach O Lerman
Journal:  Hypertension       Date:  2018-11       Impact factor: 10.190

10.  In a Phase 1a escalating clinical trial, autologous mesenchymal stem cell infusion for renovascular disease increases blood flow and the glomerular filtration rate while reducing inflammatory biomarkers and blood pressure.

Authors:  Abdelrhman Abumoawad; Ahmed Saad; Christopher M Ferguson; Alfonso Eirin; Sandra M Herrmann; LaTonya J Hickson; Busra B Goksu; Emily Bendel; Sanjay Misra; James Glockner; Allan B Dietz; Lilach O Lerman; Stephen C Textor
Journal:  Kidney Int       Date:  2019-12-13       Impact factor: 10.612
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