Literature DB >> 10725280

Vascular effects following homozygous disruption of p47(phox) : An essential component of NADPH oxidase.

E Hsich1, B H Segal, P J Pagano, F E Rey, B Paigen, J Deleonardis, R F Hoyt, S M Holland, T Finkel.   

Abstract

BACKGROUND: Evidence suggests that the vessel wall contains an oxidase similar, if not identical, to phagocytic NADPH oxidase. We tested the contribution of this specific oxidase to the progression of atherosclerosis and the regulation of blood pressure. METHODS AND
RESULTS: An examination of aortic rings from wild-type mice and mice with homozygous targeted disruptions in p47(phox) revealed that p47(phox) knockout mice had a reduction in vascular superoxide production. However, analyses of apoE -/- p47(phox)+/+ and apoE -/- p47(phox) -/- strains of mice demonstrated no significant differences in atherosclerotic lesion sizes. Similarly, analyses of wild-type and p47(phox) knockout mice revealed no differences in either basal blood pressure or the rise in blood pressure seen after the pharmacological inhibition of nitric oxide synthase.
CONCLUSIONS: NADPH oxidase contributes to basal vascular superoxide production. However, the absence of a functional oxidase does not significantly affect the progression of atherosclerosis in the standard mouse apoE -/- model, nor does it significantly influence basal blood pressure.

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Year:  2000        PMID: 10725280     DOI: 10.1161/01.cir.101.11.1234

Source DB:  PubMed          Journal:  Circulation        ISSN: 0009-7322            Impact factor:   29.690


  36 in total

1.  Out, damned dot: studies of the NADPH oxidase in atherosclerosis.

Authors:  K K Griendling; D G Harrison
Journal:  J Clin Invest       Date:  2001-11       Impact factor: 14.808

Review 2.  The Nox family of NADPH oxidases: friend or foe of the vascular system?

Authors:  Ina Takac; Katrin Schröder; Ralf P Brandes
Journal:  Curr Hypertens Rep       Date:  2012-02       Impact factor: 5.369

Review 3.  The yin/yang of superoxide dismutase mimetics: potential cardiovascular therapies?

Authors:  David S Weber; Kathy K Griendling
Journal:  Br J Pharmacol       Date:  2003-07       Impact factor: 8.739

Review 4.  Nox isoforms in vascular pathophysiology: insights from transgenic and knockout mouse models.

Authors:  Jennifer Rivera; Christopher G Sobey; Anna K Walduck; Grant R Drummond
Journal:  Redox Rep       Date:  2010       Impact factor: 4.412

Review 5.  Chronic granulomatous disease.

Authors:  Steven M Holland
Journal:  Clin Rev Allergy Immunol       Date:  2010-02       Impact factor: 8.667

Review 6.  Biochemistry, physiology, and pathophysiology of NADPH oxidases in the cardiovascular system.

Authors:  Bernard Lassègue; Alejandra San Martín; Kathy K Griendling
Journal:  Circ Res       Date:  2012-05-11       Impact factor: 17.367

Review 7.  Reactive Oxygen Species in Metabolic and Inflammatory Signaling.

Authors:  Steven J Forrester; Daniel S Kikuchi; Marina S Hernandes; Qian Xu; Kathy K Griendling
Journal:  Circ Res       Date:  2018-03-16       Impact factor: 17.367

8.  Role for Nox1 NADPH oxidase in atherosclerosis.

Authors:  Andrea L Sheehan; Samuel Carrell; Bryon Johnson; Bojana Stanic; Botond Banfi; Francis J Miller
Journal:  Atherosclerosis       Date:  2011-02-24       Impact factor: 5.162

9.  Angiotensin II induces a region-specific hyperplasia of the ascending aorta through regulation of inhibitor of differentiation 3.

Authors:  A Phillip Owens; Venkateswaran Subramanian; Jessica J Moorleghen; Zhenheng Guo; Coleen A McNamara; Lisa A Cassis; Alan Daugherty
Journal:  Circ Res       Date:  2009-12-17       Impact factor: 17.367

10.  A copy number variation in human NCF1 and its pseudogenes.

Authors:  Tiffany Brunson; Qingwei Wang; Isfahan Chambers; Qing Song
Journal:  BMC Genet       Date:  2010-02-23       Impact factor: 2.797

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