Literature DB >> 10617941

Differences in glucose-dependent insulinotrophic polypeptide hormone and hepatic lipase in subjects of southern and northern Europe: implications for postprandial lipemia.

K G Jackson1, A Zampelas, J M Knapper, H M Roche, M J Gibney, A Kafatos, B J Gould, J W Wright, C M Williams.   

Abstract

BACKGROUND: This study was an extension of a previous study that showed different lipemic responses to standard test meals in subjects from southern and northern Europe.
OBJECTIVE: The aim was to determine in 32 healthy young men from northern and southern Europe whether differences in the secretion of insulin and glucose-dependent insulinotrophic polypeptide (GIP) might explain these findings through the actions of these hormones on lipoprotein lipase.
DESIGN: We investigated in a randomized, single-blind, crossover study the effects of 2 test meals of identical macronutrient composition but different saturated fatty acid (SFA) and monounsaturated fatty acid (MUFA) contents on postprandial GIP, insulin, the ratio of incremental triacylglycerol to apolipoprotein B-48 (a marker of chylomicron size), and the activity of postheparin lipases.
RESULTS: Fasting and postprandial GIP concentrations and postheparin hepatic lipase activities were significantly higher in the southern Europeans (P < 0.001 and P < 0.02, respectively). Lipoprotein lipase activity after the SFA-rich meal was significantly higher in the northern Europeans (P < 0.01). HL activity 9 h after the SFA-rich meal and the area under the curve (AUC) for the postprandial insulin response correlated with the AUC for the postprandial GIP response [r = 0.44 (P < 0.04) and r = 0.46 (P < 0.05), respectively]. There were no significant differences in chylomicron size between the 2 groups for either meal, but when the groups were combined there was a significant difference in chylomicron size between the SFA- and MUFA-rich meals (P < 0.05), which could be due to the formation of larger chylomicrons after the MUFA-rich meal.
CONCLUSION: The significantly higher GIP and insulin responses and HL activities in southern Europeans may provide an explanation for our previous report of attenuated postprandial triacylglycerol and apolipoprotein B-48 responses in them.

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Year:  2000        PMID: 10617941     DOI: 10.1093/ajcn/71.1.13

Source DB:  PubMed          Journal:  Am J Clin Nutr        ISSN: 0002-9165            Impact factor:   7.045


  3 in total

1.  Introduction to the DISRUPT postprandial database: subjects, studies and methodologies.

Authors:  Kim G Jackson; Dave T Clarke; Peter Murray; Julie A Lovegrove; Brendan O'Malley; Anne M Minihane; Christine M Williams
Journal:  Genes Nutr       Date:  2009-09-29       Impact factor: 5.523

2.  Interaction between cholesteryl ester transfer protein and hepatic lipase encoding genes and the risk of type 2 diabetes: results from the Telde study.

Authors:  Laura López-Ríos; Francisco J Nóvoa; Ricardo Chirino; Francisco Varillas; Mauro Boronat-Cortés; Ana M Wägner
Journal:  PLoS One       Date:  2011-11-03       Impact factor: 3.240

3.  The dual glucose-dependent insulinotropic peptide and glucagon-like peptide-1 receptor agonist, tirzepatide, improves lipoprotein biomarkers associated with insulin resistance and cardiovascular risk in patients with type 2 diabetes.

Authors:  Jonathan M Wilson; Amir Nikooienejad; Deborah A Robins; William C Roell; Jeffrey S Riesmeyer; Axel Haupt; Kevin L Duffin; Marja-Riitta Taskinen; Giacomo Ruotolo
Journal:  Diabetes Obes Metab       Date:  2020-09-15       Impact factor: 6.577

  3 in total

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