Literature DB >> 10559074

The incidence, prevalence, and severity of sarcoidosis in New York City firefighters.

D J Prezant1, A Dhala, A Goldstein, D Janus, F Ortiz, T K Aldrich, K J Kelly.   

Abstract

OBJECTIVE: The etiology of sarcoidosis is unknown, but epidemiology suggests that environmental agents are a factor. Because firefighters are exposed to numerous toxins, we questioned whether sarcoidosis was increased in this cohort.
SETTING: The New York City Fire Department (FDNY), employing > 11,000 firefighters and nearly 3,000 emergency medical services (EMS) health-care workers (HCWs).
DESIGN: In 1985, FDNY initiated a surveillance program to determine the incidence, prevalence, and severity of biopsy-proven sarcoidosis in firefighters. In 1995, EMS HCWs were added as control subjects.
RESULTS: Between 1985 and 1998, 4 prior cases and 21 new cases of sarcoidosis were found in FDNY firefighters. Annual incidence proportions ranged from 0 to 43.6/100,000, and averaged 12.9/100,000. On July 1, 1998, the point prevalence was 222/100,000. For EMS HCWs, annual incidence proportions were zero. Radiographic stage 0 or stage 1 sarcoidosis was found in 19 firefighters (76%), and stage 3 was found in 1 firefighter (4%). Pulmonary function (FVC, FEV(1), and diffusing capacity for carbon monoxide) was normal in 17 firefighters (68%), and reduced to </= 65% predicted in 2 firefighters (8%). Maximum oxygen consumption (MVO(2)) was normal in 10 of 17 firefighters (59%), and reduced to 65% predicted in 3 firefighters (12%). Five of seven firefighters (71%) with abnormal MVO(2) had gas exchange abnormalities, and none had O(2) desaturation. All returned to fire fighting.
CONCLUSIONS: Annual incidence proportions and point prevalence were increased in FDNY firefighters as compared to EMS HCWs and historical controls. Radiographs and physiologic measurements demonstrated only minimal impairment.

Entities:  

Mesh:

Year:  1999        PMID: 10559074     DOI: 10.1378/chest.116.5.1183

Source DB:  PubMed          Journal:  Chest        ISSN: 0012-3692            Impact factor:   9.410


  37 in total

1.  Sarcoidosis and the occurrence of malignant diseases.

Authors:  Norbert Blank; Hanns-Martin Lorenz; Anthony D Ho; Mathias Witzens-Harig
Journal:  Rheumatol Int       Date:  2014-03-23       Impact factor: 2.631

2.  Peroxisome Proliferator-activated Receptor-γ Deficiency Exacerbates Fibrotic Response to Mycobacteria Peptide in Murine Sarcoidosis Model.

Authors:  Anagha Malur; Arjun Mohan; Robert A Barrington; Nancy Leffler; Amrita Malur; Barbara Muller-Borer; Gina Murray; Kim Kew; Chuanzhen Zhou; Josh Russell; Jacob L Jones; Christopher J Wingard; Barbara P Barna; Mary Jane Thomassen
Journal:  Am J Respir Cell Mol Biol       Date:  2019-08       Impact factor: 6.914

3.  Post-9/11 sarcoidosis in WTC-exposed firefighters and emergency medical service workers.

Authors:  Mayris P Webber; Jennifer Yip; Rachel Zeig-Owens; William Moir; Patompong Ungprasert; Cynthia S Crowson; Charles B Hall; Nadia Jaber; Michael D Weiden; Eric L Matteson; David J Prezant
Journal:  Respir Med       Date:  2017-06-07       Impact factor: 3.415

4.  Alveolar Macrophage ABCG1 Deficiency Promotes Pulmonary Granulomatous Inflammation.

Authors:  Matthew McPeek; Anagha Malur; Debra A Tokarz; Kvin Lertpiriyapong; Kymberly M Gowdy; Gina Murray; Christopher J Wingard; Michael B Fessler; Barbara P Barna; Mary Jane Thomassen
Journal:  Am J Respir Cell Mol Biol       Date:  2019-09       Impact factor: 6.914

Review 5.  How the Frequency and Phenotype of Sarcoidosis is Driven by Environmental Determinants.

Authors:  Manuel Ramos-Casals; Belchin Kostov; Pilar Brito-Zerón; Antoni Sisó-Almirall; Robert P Baughman
Journal:  Lung       Date:  2019-06-12       Impact factor: 2.584

6.  Exposure to a Mycobacterial Antigen, ESAT-6, Exacerbates Granulomatous and Fibrotic Changes in a Multiwall Carbon Nanotube Model of Chronic Pulmonary Disease.

Authors:  Anagha Malur; Barbara P Barna; Janki Patel; Matthew McPeek; Christopher J Wingard; Larry Dobbs; Mary Jane Thomassen
Journal:  J Nanomed Nanotechnol       Date:  2015-12-27

7.  Epidemiology of Sarcoidosis 1946-2013: A Population-Based Study.

Authors:  Patompong Ungprasert; Eva M Carmona; James P Utz; Jay H Ryu; Cynthia S Crowson; Eric L Matteson
Journal:  Mayo Clin Proc       Date:  2015-12-22       Impact factor: 7.616

8.  PPAR-gamma pathways attenuate pulmonary granuloma formation in a carbon nanotube induced murine model of sarcoidosis.

Authors:  Matthew McPeek; Anagha Malur; Debra A Tokarz; Gina Murray; Barbara P Barna; Mary Jane Thomassen
Journal:  Biochem Biophys Res Commun       Date:  2018-06-15       Impact factor: 3.575

Review 9.  World Trade Center Cough Syndrome and its treatment.

Authors:  David J Prezant
Journal:  Lung       Date:  2007-11-20       Impact factor: 2.584

10.  Genetic variants in mannose receptor gene (MRC1) confer susceptibility to increased risk of sarcoidosis.

Authors:  Takeshi Hattori; Satoshi Konno; Ayumu Takahashi; Akira Isada; Kaoruko Shimizu; Kenichi Shimizu; Natsuko Taniguchi; Peisong Gao; Etsuro Yamaguchi; Nobuyuki Hizawa; Shau-Ku Huang; Masaharu Nishimura
Journal:  BMC Med Genet       Date:  2010-10-28       Impact factor: 2.103

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