Literature DB >> 10522858

Osteoporosis in multiple endocrine neoplasia type 1: severity, clinical significance, relationship to primary hyperparathyroidism, and response to parathyroidectomy.

J R Burgess1, R David, T M Greenaway, V Parameswaran, J J Shepherd.   

Abstract

BACKGROUND: Sporadic primary hyperparathyroidism (PHPT) occurs most frequently in postmenopausal women. Multiple endocrine neoplasia type 1 (MEN 1) is an autosomal-dominant disease in which mild to moderate PHPT develops in most gene carriers by 20 years of age. Primary hyperparathyroidism associated with MEN 1 is typically recurrent, despite initially successful subtotal parathyroidectomy. Osteoporosis is considered a complication of sporadic PHPT and an indication for parathyroidectomy. In the setting of MEN 1, however, the relationship of bone mass to PHPT, fracture risk, and parathyroidectomy is unknown. HYPOTHESIS: Parathyroidectomy improves bone mineral density for patients with primary hyperparathyroidism in the setting of MEN 1.
DESIGN: Case series.
SETTING: Tertiary referral center. PATIENTS: Twenty-nine women with MEN 1 belonging to a single family with a history of MEN 1.
INTERVENTIONS: Parathyroidectomy. MAIN OUTCOME MEASURES: Bone mineral density (BMD) and history of skeletal fracture.
RESULTS: Osteopenia and osteoporosis were diagnosed in 41% and 45% of patients, respectively. Forty-four percent of patients with uncontrolled PHPT had severe osteopenia (T score, <-2.0) by 35 years of age. Reduction in BMD was greatest at the femoral neck. Reduced BMD was associated with an increased likelihood of skeletal fracture (P = .05). Patients with uncontrolled PHPT had lower femoral neck and lumbar spine BMDs than those in whom PHPT was controlled by parathyroidectomy (P = .005 and .02, respectively). Successful parathyroidectomy improved femoral neck and lumbar spine BMDs by a mean +/- SEM of 5.2% +/- 2.5% and 3.2% +/- 2.9%, respectively.
CONCLUSIONS: Osteoporosis is a frequent and early complication of PHPT in MEN 1. Despite difficulty in achieving a cure of PHPT in MEN 1, parathyroidectomy has an important role in the optimization of BMD for patients with MEN 1.

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Mesh:

Year:  1999        PMID: 10522858     DOI: 10.1001/archsurg.134.10.1119

Source DB:  PubMed          Journal:  Arch Surg        ISSN: 0004-0010


  29 in total

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Authors:  Maria Luisa Brandi
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Review 2.  Clinical aspects of multiple endocrine neoplasia type 1.

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3.  Causes and treatment of recurrent hyperparathyroidism after subtotal parathyroidectomy in the presence of multiple endocrine neoplasia 1.

Authors:  Maria D Balsalobre Salmeron; Jose Manuel Rodriguez Gonzalez; Joan Sancho Insenser; Joan Sancho Fornos; Albert Goday; Nuria Maria Torregrosa Perez; Antonio Rios Zambudio; Pascual Parrilla Paricio; Antonio Sitges Serra
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4.  Aortopulmonary window parathyroid gland causing primary hyperparathyroidism in men type 1 syndrome.

Authors:  Francesco Tonelli; Carlo Biagini; Francesco Giudici; Federica Cioppi; Maria Luisa Brandi
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5.  Peripheral bone mineral density in correlation to disease-related predisposing conditions in patients with multiple endocrine neoplasia type 1.

Authors:  P H Kann; D Bartsch; P Langer; J Waldmann; P Hadji; A Pfützner; J Klüsener
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6.  Hyperosmotic Stimulus Down-regulates 1alpha, 25-dihydroxyvitamin D(3)-induced Osteoclastogenesis by Suppressing the RANKL Expression in a Co-culture System.

Authors:  Yu Shun Tian; Hyun Joo Jeong; Sang-Do Lee; Seok Heui Kong; Seung-Ho Ohk; Yun-Jung Yoo; Jeong-Taeg Seo; Dong Min Shin; Byung-Wha Sohn; Syng-Ill Lee
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Review 7.  Familial parathyroid tumors: diagnosis and management.

Authors:  Peter Stålberg; Tobias Carling
Journal:  World J Surg       Date:  2009-11       Impact factor: 3.352

8.  Clinical Use of Cinacalcet in MEN1 Hyperparathyroidism.

Authors:  V J Moyes; J P Monson; S L Chew; S A Akker
Journal:  Int J Endocrinol       Date:  2010-05-26       Impact factor: 3.257

Review 9.  Multiple Endocrine Neoplasia: Genetics and Clinical Management.

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10.  [Multiple endocrine neoplasia type 1. Surgical therapy of primary hyperparathyroidism].

Authors:  P Langer; A Wild; T Schilling; C Nies; M Rothmund; D K Bartsch
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