| Literature DB >> 9797467 |
S P Hogan1, A W Mould, J M Young, M E Rothenberg, A J Ramsay, K Matthaei, I G Young, P S Foster.
Abstract
Airway inflammation is central to the pathogenesis of allergic asthma, and molecules that mediate this process obviously represent targets for therapy. In the present article, we discuss our experiments, which point to CD4+ T cells and IL-5-driven eosinophilia as potential targets for the relief of bronchial hyperreactivity in late-phase asthma.Entities:
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Year: 1998 PMID： 9797467 DOI： 10.1046/j.1440-1711.1998.00766.x
Source DB: PubMed Journal: Immunol Cell Biol ISSN： 0818-9641 Impact factor: 5.126