Literature DB >> 9749974

Urinary N-acetyl-beta-glucosaminidase as an indicator of renal dysfunction in electroplating workers.

C S Liu1, H W Kuo, J S Lai, T I Lin.   

Abstract

OBJECTIVES: To investigate chromium-induced renal dysfunction in electroplating workers.
METHODS: A cross-sectional study was used to evaluate four biochemical markers of renal function. A total of 178 workers were divided into 3 comparable groups consisting of 34 hard-chrome plating workers, 98 nickel-chrome electroplating workers. and 46 aluminum anode-oxidation workers, who represented the reference group. Ambient and biological monitoring of urinary chromium were performed to measure exposure concentrations.
RESULTS: Overall, urinary chromium concentrations were highest among hard-chrome plating workers (geometric mean 2.44 microg/g creatinine), followed by nickel-chrome electroplating workers (0.31 microg/g creatinine) and aluminum workers (0.09 microg/g creatinine). Airborne chromium concentrations were also highest in the hard-chrome plating area (geometric mean 4.20 microg/m3), followed by the nickel-chrome electroplating area (0.58 microg/m3) and the aluminum area (0.43 microg/m3). A positive correlation was found between urinary chromium and airborne concentrations (r=0.54, P < 0.01). Urinary concentrations of N-acetyl-beta-D-glucosaminidase (NAG) were also highest among hard-chrome plating workers (geometric mean 4.9 IU/g creatinine), followed by nickel-chrome workers (3.4 IU/g creatinine) and aluminum workers (2.9 IU/g creatinine). The prevalence of "elevated" NAG (>7 IU/g creatinine) was significantly highest among hard-chrome plating workers (23.5%), then among nickel-chrome workers (7.1%) and aluminum workers (8.7%). Differences in beta2-microglobulin, total protein, and microalbumin were not significant.
CONCLUSION: The author's evidence indicates that NAG is an early indicator of renal dysfunction in hard-chrome plating workers.

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Year:  1998        PMID: 9749974     DOI: 10.1007/s004200050291

Source DB:  PubMed          Journal:  Int Arch Occup Environ Health        ISSN: 0340-0131            Impact factor:   3.015


  5 in total

1.  Renal impairment caused by chronic occupational chromate exposure.

Authors:  Tiancheng Wang; Guang Jia; Ji Zhang; Yanhui Ma; Weiyue Feng; Lanzheng Liu; Ning Zhang; Lei Yan; Xiang Wang; Xueyan Zhang; Zihong Liu; Xianmei Du; Sen Zhen
Journal:  Int Arch Occup Environ Health       Date:  2010-08-18       Impact factor: 3.015

2.  Preliminary screening of the possible protective effect of Moroccan propolis against chromium-induced nephrotoxicity in animal model.

Authors:  Soukaina El-Guendouz; Soumia Zizi; Youssef Elamine; Badiaa Lyoussi
Journal:  Vet World       Date:  2020-07-14

3.  Reliability and validity of expert assessment based on airborne and urinary measures of nickel and chromium exposure in the electroplating industry.

Authors:  Yu-Cheng Chen; Joseph B Coble; Nicole C Deziel; Bu-Tian Ji; Shouzheng Xue; Wei Lu; Patricia A Stewart; Melissa C Friesen
Journal:  J Expo Sci Environ Epidemiol       Date:  2014-04-16       Impact factor: 5.563

4.  Renal tubular dysfunction in children living in the Aral Sea Region.

Authors:  K Kaneko; M Chiba; M Hashizume; O Kunii; S Sasaki; T Shimoda; Y Yamashiro; W Caypil; D Dauletbaev
Journal:  Arch Dis Child       Date:  2003-11       Impact factor: 3.791

5.  Simvastatin attenuates chromium-induced nephrotoxicity in rats.

Authors:  Zahra Goodarzi; Esmaeil Karami; Massumeh Ahmadizadeh
Journal:  J Nephropathol       Date:  2016-08-09
  5 in total

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