Literature DB >> 9726049

Multiple synchronous lung cancers and atypical adenomatous hyperplasia in Li-Fraumeni syndrome.

Y Nadav1, U Pastorino, A G Nicholson.   

Abstract

AIM: We report a patient with Li-Fraumeni syndrome who developed multiple synchronous primary lung cancers together with atypical adenomatous hyperplasia, and discuss the pathogenesis of the lesions. CASE DETAILS: A 57-year-old woman with Li-Fraumeni syndrome and a history of smoking was diagnosed as having multiple synchronous primary lung tumours, all of which were treated by surgical excision. Histological examination showed three separate well-differentiated adenocarcinomas and a single focus of atypical adenomatous hyperplasia. A constitutional mutation of the p53 gene had been identified, both in this patient and other members of her family.
CONCLUSION: Multiple synchronous primary lung tumours have not been reported before in association with this syndrome. With both smoking and Li-Fraumeni syndrome known to be associated with mutations in the p53 gene, a cumulative effect is suggested in this patient.

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Year:  1998        PMID: 9726049     DOI: 10.1046/j.1365-2559.1998.00439.x

Source DB:  PubMed          Journal:  Histopathology        ISSN: 0309-0167            Impact factor:   5.087


  3 in total

Review 1.  Pulmonary preinvasive neoplasia.

Authors:  K M Kerr
Journal:  J Clin Pathol       Date:  2001-04       Impact factor: 3.411

2.  Lung adenocarcinoma associated with familial adenomatous polyposis. Clear cell carcinoma with beta-catenin accumulation accompanied by atypical adenomatous hyperplasia.

Authors:  Akiteru Goto; Jun Nakajima; Kei Hara; Toshiro Niki; Masashi Fukayama
Journal:  Virchows Arch       Date:  2004-11-16       Impact factor: 4.064

3.  The association between atypical adenomatous hyperplasia and primary lung cancer.

Authors:  A D Chapman; K M Kerr
Journal:  Br J Cancer       Date:  2000-09       Impact factor: 7.640

  3 in total

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