Literature DB >> 9713429

Oxidative stress and coronary artery disease.

D Harrison1.   

Abstract

Over the past 10 years, research into oxidative processes in the vascular wall has taken two parallel tracks that are beginning to merge. One of the tracks concerns the oxidation of lipids, and the other is the impact of oxidative reactions on nitric oxide biology. This update focuses on data implicating oxidative inactivation of nitric oxide in atherosclerosis and hypercholesterolemia.

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Year:  1998        PMID： 9713429

Source DB:  PubMed          Journal:  Can J Cardiol        ISSN： 0828-282X            Impact factor:   5.223


  3 in total

1.  Systemic elevations of free radical oxidation products of arachidonic acid are associated with angiographic evidence of coronary artery disease.

Authors:  Mehdi H Shishehbor; Renliang Zhang; Hector Medina; Marie-Luise Brennan; Danielle M Brennan; Stephen G Ellis; Eric J Topol; Stanley L Hazen
Journal:  Free Radic Biol Med       Date:  2006-09-08       Impact factor: 7.376

2.  Systemically administered tempol reduces neuronal activity in paraventricular nucleus of hypothalamus and rostral ventrolateral medulla in rats.

Authors:  Shun-Guang Wei; Zhi-Hua Zhang; Yang Yu; Robert B Felder
Journal:  J Hypertens       Date:  2009-03       Impact factor: 4.844

3.  Apolipoprotein A-I mimetic peptides prevent atherosclerosis development and reduce plaque inflammation in a murine model of diabetes.

Authors:  Cecilia Morgantini; Satoshi Imaizumi; Victor Grijalva; Mohamad Navab; Alan M Fogelman; Srinivasa T Reddy
Journal:  Diabetes       Date:  2010-09-08       Impact factor: 9.461
  3 in total

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