Literature DB >> 9671583

LIN-12 protein expression and localization during vulval development in C. elegans.

D Levitan1, I Greenwald.   

Abstract

We have used a LIN-12::GFP fusion protein to examine LIN-12 accumulation during cell fate decisions important for vulval development. During the naturally variable anchor cell (AC)/ventral uterine precursor cell (VU) decision of the somatic gonad, a transcription-based feedback mechanism biases two equivalent cells so that one becomes the AC while the other becomes a VU. LIN-12::GFP accumulation reflects lin-12 transcription: LIN-12::GFP is initially present in both cells, but disappears from the presumptive AC and becomes restricted to the presumptive VU. During vulval precursor cell (VPC) fate determination, six equipotential cells uniformly transcribe lin-12 and have invariant fates that are specified by multiple cell-cell interactions. The pattern of LIN-12::GFP accumulation in VPCs and in the VPC lineages is dynamic and does not always reflect lin-12 transcription. In particular, LIN-12::GFP is expressed initially in all six VPCs, but appears to be reduced specifically in P6.p as a consequence of the activation of the Ras pathway by an EGF-like inductive signal from the AC. We propose that downregulation of LIN-12 stability or translation in response to inductive signalling helps impose a bias on lateral signalling and contributes to the invariant pattern of VPC fates.

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Year:  1998        PMID: 9671583     DOI: 10.1242/dev.125.16.3101

Source DB:  PubMed          Journal:  Development        ISSN: 0950-1991            Impact factor:   6.868


  25 in total

1.  Intercellular coupling amplifies fate segregation during Caenorhabditis elegans vulval development.

Authors:  Claudiu A Giurumescu; Paul W Sternberg; Anand R Asthagiri
Journal:  Proc Natl Acad Sci U S A       Date:  2006-01-23       Impact factor: 11.205

2.  Integration of EGFR and LIN-12/Notch Signaling by LIN-1/Elk1, the Cdk8 Kinase Module, and SUR-2/Med23 in Vulval Precursor Cell Fate Patterning in Caenorhabditis elegans.

Authors:  Ryan S Underwood; Yuting Deng; Iva Greenwald
Journal:  Genetics       Date:  2017-09-27       Impact factor: 4.562

3.  Ras effector switching promotes divergent cell fates in C. elegans vulval patterning.

Authors:  Tanya P Zand; David J Reiner; Channing J Der
Journal:  Dev Cell       Date:  2011-01-18       Impact factor: 12.270

4.  SEL-5, a serine/threonine kinase that facilitates lin-12 activity in Caenorhabditis elegans.

Authors:  H Fares; I Greenwald
Journal:  Genetics       Date:  1999-12       Impact factor: 4.562

Review 5.  Morphogenesis of the caenorhabditis elegans vulva.

Authors:  Adam J Schindler; David R Sherwood
Journal:  Wiley Interdiscip Rev Dev Biol       Date:  2013 Jan-Feb       Impact factor: 5.814

6.  Suppressors of the egg-laying defective phenotype of sel-12 presenilin mutants implicate the CoREST corepressor complex in LIN-12/Notch signaling in C. elegans.

Authors:  Sophie Jarriault; Iva Greenwald
Journal:  Genes Dev       Date:  2002-10-15       Impact factor: 11.361

7.  The C. elegans homolog of the mammalian tumor suppressor Dep-1/Scc1 inhibits EGFR signaling to regulate binary cell fate decisions.

Authors:  Thomas A Berset; Erika Fröhli Hoier; Alex Hajnal
Journal:  Genes Dev       Date:  2005-05-18       Impact factor: 11.361

8.  sel-11 and cdc-42, two negative modulators of LIN-12/Notch activity in C. elegans.

Authors:  Min Sung Choi; Andrew S Yoo; Iva Greenwald
Journal:  PLoS One       Date:  2010-07-29       Impact factor: 3.240

9.  Control of cell-fate plasticity and maintenance of multipotency by DAF-16/FoxO in quiescent Caenorhabditis elegans.

Authors:  Xantha Karp; Iva Greenwald
Journal:  Proc Natl Acad Sci U S A       Date:  2013-01-22       Impact factor: 11.205

10.  Dorsoventral patterning of the C. elegans postembryonic mesoderm requires both LIN-12/Notch and TGFbeta signaling.

Authors:  Marisa L Foehr; Jun Liu
Journal:  Dev Biol       Date:  2007-10-25       Impact factor: 3.582

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