Literature DB >> 9670323

Regulation of the cell cycle by the Rb tumor suppressor family.

M E Ewen1.   

Abstract

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Year:  1998        PMID： 9670323     DOI： 10.1007/978-3-540-69686-5_7

Source DB:  PubMed          Journal:  Results Probl Cell Differ        ISSN： 0080-1844


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  5 in total

1.  Discovery of Dinaciclib (SCH 727965): A Potent and Selective Inhibitor of Cyclin-Dependent Kinases.

Authors:  Kamil Paruch; Michael P Dwyer; Carmen Alvarez; Courtney Brown; Tin-Yau Chan; Ronald J Doll; Kerry Keertikar; Chad Knutson; Brian McKittrick; Jocelyn Rivera; Randall Rossman; Greg Tucker; Thierry Fischmann; Alan Hruza; Vincent Madison; Amin A Nomeir; Yaolin Wang; Paul Kirschmeier; Emma Lees; David Parry; Nicole Sgambellone; Wolfgang Seghezzi; Lesley Schultz; Frances Shanahan; Derek Wiswell; Xiaoying Xu; Quiao Zhou; Ray A James; Vidyadhar M Paradkar; Haengsoon Park; Laura R Rokosz; Tara M Stauffer; Timothy J Guzi
Journal:  ACS Med Chem Lett       Date:  2010-05-17       Impact factor: 4.345

2.  A high degree of chromosomal instability at 13q14 in cutaneous squamous cell carcinomas: indication for a role of a tumour suppressor gene other than Rb.

Authors:  D P O'Connor; E W Kay; M Leader; G M Murphy; G J Atkins; M J Mabruk
Journal:  Mol Pathol       Date:  2001-06

3.  The retinoblastoma protein is linked to the activation of Ras.

Authors:  K Y Lee; M H Ladha; C McMahon; M E Ewen
Journal:  Mol Cell Biol       Date:  1999-11       Impact factor: 4.272

4.  QM-FISH analysis of the genes involved in the G1/S checkpoint signaling pathway in triple-negative breast cancer.

Authors:  Sheng Zhang; Yingbo Shao; Guofang Hou; Jingchao Bai; Weiping Yuan; Linping Hu; Tao Cheng; Anders Zetterberg; Jin Zhang
Journal:  Tumour Biol       Date:  2013-10-05

5.  FGF signaling targets the pRb-related p107 and p130 proteins to induce chondrocyte growth arrest.

Authors:  Emmanuel Laplantine; Ferdinand Rossi; Malika Sahni; Claudio Basilico; David Cobrinik
Journal:  J Cell Biol       Date:  2002-08-12       Impact factor: 10.539
  5 in total

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