Literature DB >> 9634084

Roles of vaccinia virus EEV-specific proteins in intracellular actin tail formation and low pH-induced cell-cell fusion.

C M Sanderson1, F Frischknecht, M Way, M Hollinshead, G L Smith.   

Abstract

During vaccinia virus (VV) morphogenesis intracellular mature virus (IMV) is wrapped by two additional membranes to form intracellular enveloped virus (IEV). IEV particles can nucleate the formation of actin tails which aid movement of IEVs to the cell surface where the outer IEV membrane fuses with the plasma membrane forming cell-associated enveloped virus (CEV) which remains attached to the cell, or extracellular enveloped virus (EEV) which is shed from the cell. In this report, we have used a collection of VV mutants lacking individual EEV-specific proteins to compare the roles of these proteins in the formation of IEV and IEV-associated actin tails and fusion of infected cells after a low pH shock. Data presented here show that p45-50 (A36R) is not required for IEV formation or for acid-induced cell-cell fusion, but is required for formation of IEV-associated actin tails. In contrast, gp86 (A56R), the virus haemagglutinin, is not required for formation of either IEV or IEV-associated actin tails. Data presented also confirm that p37 (gene F13L), gp42 (B5R) and gp22-24 (A34R) are needed for formation of IEV-associated actin tails and for cell-cell fusion after low pH shock. The phenotypes of these mutants were not affected by the host cell type as similar results were obtained in a range of different cells. Lastly, comparisons of the phenotypes of VV strains Western Reserve, deltaA34R and deltaA36R demonstrate that actin tails are not required for low pH-induced cell-cell fusion.

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Year:  1998        PMID: 9634084     DOI: 10.1099/0022-1317-79-6-1415

Source DB:  PubMed          Journal:  J Gen Virol        ISSN: 0022-1317            Impact factor:   3.891


  60 in total

1.  Golgi network targeting and plasma membrane internalization signals in vaccinia virus B5R envelope protein.

Authors:  B M Ward; B Moss
Journal:  J Virol       Date:  2000-04       Impact factor: 5.103

2.  Regulation of vaccinia virus morphogenesis: phosphorylation of the A14L and A17L membrane proteins and C-terminal truncation of the A17L protein are dependent on the F10L kinase.

Authors:  T Betakova; E J Wolffe; B Moss
Journal:  J Virol       Date:  1999-05       Impact factor: 5.103

3.  Vaccinia virus F12L protein is required for actin tail formation, normal plaque size, and virulence.

Authors:  W H Zhang; D Wilcock; G L Smith
Journal:  J Virol       Date:  2000-12       Impact factor: 5.103

4.  Effects of deletion or stringent repression of the H3L envelope gene on vaccinia virus replication.

Authors:  F G da Fonseca; E J Wolffe; A Weisberg; B Moss
Journal:  J Virol       Date:  2000-08       Impact factor: 5.103

Review 5.  Actin-based motility of intracellular microbial pathogens.

Authors:  M B Goldberg
Journal:  Microbiol Mol Biol Rev       Date:  2001-12       Impact factor: 11.056

Review 6.  Directed egress of animal viruses promotes cell-to-cell spread.

Authors:  David C Johnson; Mary T Huber
Journal:  J Virol       Date:  2002-01       Impact factor: 5.103

7.  Visualization of intracellular movement of vaccinia virus virions containing a green fluorescent protein-B5R membrane protein chimera.

Authors:  B M Ward; B Moss
Journal:  J Virol       Date:  2001-05       Impact factor: 5.103

8.  The vaccinia virus A9L gene encodes a membrane protein required for an early step in virion morphogenesis.

Authors:  W W Yeh; B Moss; E J Wolffe
Journal:  J Virol       Date:  2000-10       Impact factor: 5.103

9.  Identification of second-site mutations that enhance release and spread of vaccinia virus.

Authors:  Ehud Katz; Elizabeth Wolffe; Bernard Moss
Journal:  J Virol       Date:  2002-11       Impact factor: 5.103

10.  Mutations in the vaccinia virus A33R and B5R envelope proteins that enhance release of extracellular virions and eliminate formation of actin-containing microvilli without preventing tyrosine phosphorylation of the A36R protein.

Authors:  Ehud Katz; Brian M Ward; Andrea S Weisberg; Bernard Moss
Journal:  J Virol       Date:  2003-11       Impact factor: 5.103

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