Literature DB >> 9622749

Anti-inflammatory benefits of tilmicosin in calves with Pasteurella haemolytica-infected lungs.

A C Chin1, D W Morck, J K Merrill, H Ceri, M E Olson, R R Read, P Dick, A G Buret.   

Abstract

OBJECTIVES: To determine whether tilmicosin alters neutrophil infiltration or function, induces neutrophil apoptosis, and affects accumulation of leukotriene B4 (LTB4) or tumor necrosis factor-alpha (TNF-alpha) in lungs of calves experimentally infected with Pasteurella haemolytica. ANIMALS: 12 weight-ranked Holstein calves. PROCEDURE: Calves were given 25% propylene glycol vehicle (n = 5) or tilmicosin (10 mg/kg of body weight; n = 6) subcutaneously, 18 hours and 15 minutes before intratracheal infection with 2 x 10(8) P haemolytica organisms. Two unmanipulated calves served as controls in some experiments. Rectal temperatures were recorded 15 minutes before, and at 3-hour intervals after infection for 24 hours. Samples obtained from bronchoalveolar lavage performed 3 and 24 hours after infection were used to assess colonization by P haemolytica, and neutrophil infiltration. Neutrophil phagocytosis of P haemolytica, membrane leakage as determined by trypan blue exclusion, oxidative function as determined by nitro blue tetrazolium reduction, and apoptosis, using electron microscopy and DNA fragmentation ELISA, were determined. SOluble TNF-alpha and LTB4 were measured from supernatants from bronchoalveolar lavage samples, using ELISA.
RESULTS: Treatment with tilmicosin resulted in significant (P < 0.05) clearance of P haemolytica and neutrophil apoptosis at 3 hours, and decreased concentration of LTB4 at 24 hours. Rectal temperatures, neutrophil infiltration, phagocytosis, oxidative functions, membrane leakage, and soluble TNF-alpha concentrations were not significantly affected by tilmicosin.
CONCLUSION: Tilmicosin effectively controlled P haemolytica infection, induced neutrophil apoptosis, reduced pulmonary inflammation, and did not affect neutrophil infiltration or function. CLINICAL RELEVANCE: By inducing neutrophil apoptosis, tilmicosin prevents further amplification of inflammatory injury in P haemolytica-infected lungs.

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Year:  1998        PMID: 9622749

Source DB:  PubMed          Journal:  Am J Vet Res        ISSN: 0002-9645            Impact factor:   1.156


  10 in total

Review 1.  Immuno-modulation and anti-inflammatory benefits of antibiotics: the example of tilmicosin.

Authors:  André G Buret
Journal:  Can J Vet Res       Date:  2010-01       Impact factor: 1.310

2.  Kinetics and intrapulmonary disposition of tilmicosin after single and repeated oral bolus administrations to rabbits.

Authors:  G Gallina; L Lucatello; I Drigo; M Cocchi; S Scandurra; F Agnoletti; C Montesissa
Journal:  Vet Res Commun       Date:  2010-06       Impact factor: 2.459

3.  Bovine polymorphonuclear neutrophil-mediated phagocytosis and an immunoglobulin G2 protease produced by Porphyromonas levii.

Authors:  D A Lobb; H J Loeman; D G Sparrow; D W Morck
Journal:  Can J Vet Res       Date:  1999-04       Impact factor: 1.310

4.  Direct and indirect anti-inflammatory effects of tulathromycin in bovine macrophages: inhibition of CXCL-8 secretion, induction of apoptosis, and promotion of efferocytosis.

Authors:  Carrie D Fischer; Jennifer K Beatty; Stephanie C Duquette; Douglas W Morck; Merlyn J Lucas; André G Buret
Journal:  Antimicrob Agents Chemother       Date:  2013-01-07       Impact factor: 5.191

5.  Tilmicosin induces apoptosis in bovine peripheral neutrophils in the presence or in the absence of Pasteurella haemolytica and promotes neutrophil phagocytosis by macrophages.

Authors:  A C Chin; W D Lee; K A Murrin; D W Morck; J K Merrill; P Dick; A G Buret
Journal:  Antimicrob Agents Chemother       Date:  2000-09       Impact factor: 5.191

6.  Tilmicosin does not inhibit interleukin-8 gene expression in the bovine lung experimentally infected with Mannheimia (Pasteurella) haemolytica.

Authors:  S Goubau; D W Morck; A Buret
Journal:  Can J Vet Res       Date:  2000-10       Impact factor: 1.310

7.  Anti-Inflammatory benefits of antibiotic-induced neutrophil apoptosis: tulathromycin induces caspase-3-dependent neutrophil programmed cell death and inhibits NF-kappaB signaling and CXCL8 transcription.

Authors:  Carrie D Fischer; Jennifer K Beatty; Cheryl G Zvaigzne; Douglas W Morck; Merlyn J Lucas; A G Buret
Journal:  Antimicrob Agents Chemother       Date:  2010-10-18       Impact factor: 5.191

8.  Tulathromycin exerts proresolving effects in bovine neutrophils by inhibiting phospholipases and altering leukotriene B4, prostaglandin E2, and lipoxin A4 production.

Authors:  Carrie D Fischer; Stephanie C Duquette; Bernard S Renaux; Troy D Feener; Douglas W Morck; Morley D Hollenberg; Merlyn J Lucas; Andre G Buret
Journal:  Antimicrob Agents Chemother       Date:  2014-05-12       Impact factor: 5.191

9.  Immuno-modulating properties of Tulathromycin in porcine monocyte-derived macrophages infected with porcine reproductive and respiratory syndrome virus.

Authors:  D Desmonts de Lamache; R Moges; A Siddiq; T Allain; T D Feener; G P Muench; N McKenna; R M Yates; A G Buret
Journal:  PLoS One       Date:  2019-08-23       Impact factor: 3.240

Review 10.  The immunology of the bovine respiratory disease complex.

Authors:  J A Ellis
Journal:  Vet Clin North Am Food Anim Pract       Date:  2001-11       Impact factor: 3.357

  10 in total

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