Literature DB >> 9328968

Proteinuria and tubulointerstitial injury.

L Chen1, Y Wang, Y C Tay, D C Harris.   

Abstract

These studies have demonstrated pathways whereby one urinary protein, holotransferrin, may alter proximal tubular cell function and cause tubular cytotoxicity, and at least two urinary proteins, albumin and transferrin, may mediate the development of interstitial inflammation in proteinuric renal disease.

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Mesh:

Year:  1997        PMID: 9328968

Source DB:  PubMed          Journal:  Kidney Int Suppl        ISSN: 0098-6577            Impact factor:   10.545


  3 in total

1.  Proteinuria, but Not eGFR, Predicts Stroke Risk in Chronic Kidney Disease: Chronic Renal Insufficiency Cohort Study.

Authors:  Danielle K Sandsmark; Steven R Messé; Xiaoming Zhang; Jason Roy; Lisa Nessel; Lotuce Lee Hamm; Jiang He; Edward J Horwitz; Bernard G Jaar; Radhakrishna R Kallem; John W Kusek; Emile R Mohler; Anna Porter; Stephen L Seliger; Stephen M Sozio; Raymond R Townsend; Harold I Feldman; Scott E Kasner
Journal:  Stroke       Date:  2015-06-30       Impact factor: 7.914

Review 2.  Albuminuria Is an Appropriate Therapeutic Target in Patients with CKD: The Pro View.

Authors:  Hiddo J Lambers Heerspink; Ron T Gansevoort
Journal:  Clin J Am Soc Nephrol       Date:  2015-04-17       Impact factor: 8.237

3.  A copper(II)-selective chelator ameliorates diabetes-evoked renal fibrosis and albuminuria, and suppresses pathogenic TGF-beta activation in the kidneys of rats used as a model of diabetes.

Authors:  D Gong; J Lu; X Chen; S Reddy; D J Crossman; S Glyn-Jones; Y-S Choong; J Kennedy; B Barry; S Zhang; Y-K Chan; K Ruggiero; A R J Phillips; G J S Cooper
Journal:  Diabetologia       Date:  2008-07-18       Impact factor: 10.122

  3 in total

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